نتایج جستجو برای: 1b

تعداد نتایج: 23152  

Journal: :Organic & biomolecular chemistry 2014
Shah Jaimin Balkrishna Shailesh Kumar Gajendra Kumar Azad Bhagat Singh Bhakuni Piyush Panini Navjeet Ahalawat Raghuvir Singh Tomar Michael R Detty Sangit Kumar

The reaction of KSeO(t)Bu with 2-iodo-arylbenzamides gave benzamide ring-substituted, quinine-derived isoselenazolones 1b–1d. The reaction of PhSH with ortho-methyl-substituted isoselenazolone 1b gave selenol 3b, which is oxidized by H2O2 to regenerate 1b. Isoselenazolone 1b shows a high rate (0.33 × 103 μM min(−1)) of oxidation of PhSH with H2O2, which is ∼103-fold more active than ebselen (1a...

2012
Eun Ju Cho Su Hyeon Jeong Byung Hoon Han Sang Uk Lee Byung Chul Yun Eun Taek Park

BACKGROUND/AIMS There is some controversy regarding whether or not hepatitis C virus (HCV) subtype 1b is more influential than non-1b subtypes on the progression of chronic hepatitis (CH) C to liver cirrhosis (LC) and hepatocellular carcinoma (HCC). METHODS We retrospectively analyzed 823 patients with chronic HCV infection, including 443 CH patients, 264 LC patients, and 116 HCC patients, wh...

Journal: :Current Biology 2004
Atsushi Suzuki Maki Hirata Katsusi Kamimura Rika Maniwa Tomoyuki Yamanaka Keiko Mizuno Masaru Kishikawa Hiroko Hirose Yoshiko Amano Natsuko Izumi Yoshihiro Miwa Shigeo Ohno

BACKGROUND aPKC and PAR-1 are required for cell polarity in various contexts. In mammalian epithelial cells, aPKC localizes at tight junctions (TJs) and plays an indispensable role in the development of asymmetric intercellular junctions essential for the establishment and maintenance of apicobasal polarity. On the other hand, one of the mammalian PAR-1 kinases, PAR-1b/EMK1/MARK2, localizes to ...

2000
SEAN JEDRZKIEWICZ HIDETOSHI NAKAMURA ERIC S. SILVERMAN ANDREW D. LUSTER NARESH MANSHARAMANI KWANG HO IN GEN TAMURA CRAIG M. LILLY Hidetoshi Nakamura Eric S. Silverman Andrew D. Luster Naresh Mansharamani Kwang Ho

Jedrzkiewicz, Sean, Hidetoshi Nakamura, Eric S. Silverman, Andrew D. Luster, Naresh Mansharamani, Kwang Ho In, Gen Tamura, and Craig M. Lilly. IL-1b induces eotaxin gene transcription in A549 airway epithelial cells through NF-kB. Am J Physiol Lung Cell Mol Physiol 279: L1058–L1065, 2000.—Eotaxin is an asthma-related C-C chemokine that is produced in response to interleukin-1b (IL-1b). We detec...

Journal: :Journal of Social and Political Psychology 2023

Much recent work has focused on Americans’ positive and negative feelings toward their own opposing political parties. However, there is neither a consensus how to model such partisan affect, nor detailed understanding of its consequences for participation. This addressed these two gaps by first, empirically examining many dimensions best characteriz...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2005
Kazuhiko Nakagawa Yuji Noguchi Akiko Uenaka Shuichiro Sato Hideo Okumura Motoyuki Tanaka Michihide Shimono Ali Mohamed Ali Eldib Toshiro Ono Nobuya Ohara Tadashi Yoshino Kazuki Yamashita Tsukasa Tsunoda Motoi Aoe Nobuyoshi Shimizu Eiichi Nakayama

PURPOSE XAGE-1 was originally identified by the search for PAGE/GAGE-related genes using expressed sequence tag database and was shown to exhibit characteristics of cancer/testis-like antigens. Four transcript variants XAGE-1a, XAGE-1b, XAGE-1c, and XAGE-1d have been identified thus far. We recently identified XAGE-1b as a dominant antigen recognized by sera from lung adenocarcinoma patients. W...

Journal: :Drug metabolism and disposition: the biological fate of chemicals 2009
Lin Zhou Kari Schmidt Frederick R Nelson Veronica Zelesky Matthew D Troutman Bo Feng

The role of breast cancer resistance protein (Bcrp) and the combined activities of Bcrp and P-glycoprotein (P-gp, Mdr1a/1b) in limiting the brain penetration of drugs at the blood-brain barrier (BBB) were investigated using wild-type FVB, Mdr1a/1b(-/-), (-/-), Bcrp(-/-), and Mdr1a/1b(-/-), (-/-)Bcrp(-/-) mice. Four drugs, flavopiridol, imatinib mesylate (Gleevec), PF-407288, and prazosin, with ...

ژورنال: :مجله پزشکی ارومیه 0
مهشید محمدیان mahshid mohamadian , hamadan university of medical sciencesدانشگاه علوم پزشکی همدانسازمان اصلی تایید شده: دانشگاه علوم پزشکی همدان (hamadan university of medical sciences) محمدتقی گودرزی mohammad taghi goodarzi research center for molecular medicine, hamadan university of medical sciences hamadan iran, tel: +988138380462مرکز تحقیقات پزشکی موکولی دانشگاه علوم پزشکی همدان، همدان، ایران، تلفن: 08138380462سازمان اصلی تایید شده: دانشگاه علوم پزشکی همدان (hamadan university of medical sciences) مسعود سعیدی جم massoud saidijam , hamadan university of medical sciencesدانشگاه علوم پزشکی همدانسازمان اصلی تایید شده: دانشگاه علوم پزشکی همدان (hamadan university of medical sciences) جمشید کریمی jamshid karimi , hamadan university of medical sciencesدانشگاه علوم پزشکی همدانسازمان اصلی تایید شده: دانشگاه علوم پزشکی همدان (hamadan university of medical sciences) شیوا برزوئی , shiva borzouei , hamadan university of medical sciencesدانشگاه علوم پزشکی همدانسازمان اصلی تایید شده: دانشگاه علوم پزشکی همدان (hamadan university of medical sciences) علیرضا سلطانیان alireza soltanian , hamadan university of medical sciencesدانشگاه علوم پزشکی همدانسازمان اصلی تایید شده: دانشگاه علوم پزشکی همدان (hamadan university of medical sciences) مرضیه شرف بیانی

پیش زمینه و هدف: سندرم متابولیک فاکتور خطر بالقوه ای برای اختلالات قلبی عروقی و آترواسکلروز است، که بواسطه برخی تغییرات فیزیولوژیک و متابولیک ازجمله افزایش تری گلیسیرید، کاهش hdl-c، افزایش ldl-c، افزایش fbs مشخص می گردد. کلستریل استر ترانسفر پروتئین (cetp)، انتقال لیپیدهای خنثی و فسفولیپیدها را بین لیپوپروتئین ها، کاتالیز می نماید. بنابراین، این پروتئین احتمالاً می تواند در تعدیل میزان پلاسمایی ...

Journal: :Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2007
Toshiro Sugimoto Masayoshi Sakaguchi Nobuhiro Ogawa Naoko Deji Takashi Uzu Yoshihiko Nishio Yutaka Eguchi Atsunori Kashiwagi

HNF-1b mutations. Most importantly, patients with an HNF-1b mutation may also be at risk of developing renal cell carcinomas, since recent evidence suggests that HNF-1b is a tumour suppressor gene [5]. In conclusion, patients with unexplained nephropathy and/or renal cysts should be routinely screened for an HNF-1b mutation in order to differentiate the RCAD syndrome from diabetic nephropathy. ...

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