نتایج جستجو برای: allergic inflammation

تعداد نتایج: 202604  

Journal: :Journal of Allergy and Clinical Immunology 2019

Journal: :Journal of Clinical Investigation 2020

2016
Fernanda F. Cruz Zachary D. Borg Meagan Goodwin Amy L. Coffey Darcy E. Wagner Patricia R.M. Rocco Daniel J. Weiss

UNLABELLED Systemic administration of bone marrow-derived mononuclear cells (BMDMCs) or bone marrow-derived mesenchymal stromal cells (MSCs) reduces inflammation and airway hyperresponsiveness (AHR) in a murine model of Th2-mediated eosinophilic allergic airway inflammation. However, since BMDMCs are a heterogeneous population that includes MSCs, it is unclear whether the MSCs alone are respons...

2008

1 allergic diseases and cysteinyl leukotriene (lt) antagonists Allergic diseases include a variety of different illnesses (rhinitis, conjunctivitis, asthma, urticaria, and dermatitis) in which the physiological and pathological basis is the release of chemical mediators such as histamine; platelet-activating factor; metabolites of arachidonic acid; and chemotactic factors from mastocytes, basop...

Journal: :American Journal of Respiratory Cell and Molecular Biology 2013

2015
Franca Puccio R Rojas I Mosquera D Cifarrelli A Hernández M Lizarralde G Peña L Jaua C Mendoza R Reyes

Background The association with Parkinson’s disease is increased with allergic rhinitis. Increasing evidence supports an important role of central and peripheral inflammation in driving Parkinson’s (PD) initiation and progression. The link between peripheral inflammation and neurodegeneration in PD patients has been revealed in several clinical reports. In addition, gut inflammation mediated by...

Journal: :Journal of immunology 2005
R Stokes Peebles Koichi Hashimoto James R Sheller Martin L Moore Jason D Morrow Shaoquan Ji Jack A Elias Kasia Goleniewska Jamye O'neal Daphne B Mitchell Barney S Graham Weisong Zhou

Cyclooxygenase (COX) inhibition during allergic sensitization and allergen airway challenge results in augmented allergic inflammation. We hypothesized that this increase in allergic inflammation was dependent on increased generation of leukotrienes that results from COX inhibition, as leukotrienes are important proinflammatory mediators of allergic disease. To test this hypothesis, we allergic...

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