نتایج جستجو برای: c6 glioma cells

تعداد نتایج: 1414193  

2006
Takanori Ohnishi Hirotaka Matsumura Shuichi Izumoto Shoju Hiraga Toru Hayakawa

A novel model of glioma cell invasion was established by using an organotypic culture of rat brains. Brain slices prepared from 2-day-old rat neonates were maintained in a culture at the interface between air and the culture medium. The slices were placed on double-layered membranes consisting of a polycarbonate membrane with 8-/im pores and a mem brane with 0.4-fim pores. The organotypic cytoa...

Journal: :Neoplasia 2005
Cristina Trejo-Solís Guadalupe Palencia Sergio Zúñiga Andrea Rodríguez-Ropon Laura Osorio-Rico Sanchez Torres Luvia Isabel Gracia-Mora Lucrecia Marquez-Rosado Aurora Sánchez Miguel E Moreno-García Arturo Cruz María Elena Bravo-Gómez Lena Ruiz-Ramírez Sara Rodríguez-Enriquez Julio Sotelo

In this work, we investigated the effects of Casiopeina II-gly (Cas IIgly)--a new copper compound exhibiting antineoplastic activity--on glioma C6 cells under both in vitro and in vivo conditions, as an approach to identify potential therapeutic agents against malignant glioma. The exposure of C6 cells to Cas IIgly significantly inhibited cell proliferation, increased reactive oxygen species (R...

2015
Qian Ma Ying Yang Duiping Feng Shuai Zheng Ran Meng Pengyan Fa Chunjuan Zhao Hua Liu Ran Song Tao Tao Longyan Yang Jie Dai Songlin Wang Wen G. Jiang Junqi He

Gliomas are the most common primary brain malignancies and are associated with a poor prognosis. Here, we showed that the PDZ domain-containing protein membrane-associated guanylate kinase inverted 3 (MAGI3) was downregulated at the both mRNA and protein levels in human glioma samples. MAGI3 inhibited proliferation, migration, and cell cycle progression of glioma cells in its overexpression and...

Journal: :The Journal of pharmacology and experimental therapeutics 2006
Yun-Sik Lee Jie Wan Bong-Jo Kim Myung-Ae Bae Byoung J Song

We previously reported that acetaminophen (APAP, 4-hydroxyacetanilide) caused apoptosis of C6 glioma cells. Therefore, we hypothesized that the level of p53, which usually stimulates apoptosis, might be increased after APAP exposure. However, APAP exposure for 24 h markedly decreased the p53 content and its downstream target p21 in a concentration-dependent manner. Reduction of p53 was not acco...

2012
Dan Li Ke Yang Jie-Si Li Xi-Yu Ke Yu Duan Ruo Du Ping Song Ke-Fu Yu Wei Ren Dan Huang Xing-Huo Li Xin Hu Xuan Zhang Qiang Zhang

BACKGROUND Considering the observations that linoleic acid conjugated with paclitaxel (CLA-PTX) possesses antitumor activity against brain tumors, is able to cross the blood-brain barrier, but has poor water solubility, the purpose of this study was to prepare a novel CLA-PTX microemulsion and evaluate its activity against brain tumors in vitro and in vivo. METHODS The in vitro cytotoxicity o...

2014
LING LONG WEIWEI WANG XIA-DONG CAI DU CHENG XINTAO SHUAI YING PENG

Resistance to chemotherapy and the side effects of anticancer drugs are the major obstacles for glioma treatment. The aim of the present study was to develop a novel approach for the treatment of gliomas that improved the therapeutic effect; the anticancer drug, doxorubicin (DOX), was combined with short interfering (si)RNA and monomethoxy polyethylene glycol polyethylenimine superparamagnetic ...

Journal: :Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research 1994
S L Bond J F Bechberger N K Khoo C C Naus

C6 glioma cells do not express the gap junction protein connexin32 or its corresponding mRNA. Very low levels of connexin43 protein and mRNA, as well as weak intercellular coupling, have been detected. Studies investigating the role of gap junctions in cell proliferation and tumorigenesis have shown that C6 cells transfected with connexin43 have increased levels of intercellular coupling and re...

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