نتایج جستجو برای: carfilzomib

تعداد نتایج: 778  

Journal: :Blood 2014
Sara Bringhen Maria Teresa Petrucci Alessandra Larocca Concetta Conticello Davide Rossi Valeria Magarotto Pellegrino Musto Luana Boccadifuoco Massimo Offidani Paola Omedé Fabiana Gentilini Giovannino Ciccone Giulia Benevolo Mariella Genuardi Vittorio Montefusco Stefania Oliva Tommaso Caravita Paola Tacchetti Mario Boccadoro Pieter Sonneveld Antonio Palumbo

This multicenter, open-label phase 2 trial determined the safety and efficacy of carfilzomib, a novel and irreversible proteasome inhibitor, in combination with cyclophosphamide and dexamethasone (CCyd) in patients with newly diagnosed multiple myeloma (NDMM) ≥65 years of age or who were ineligible for autologous stem cell transplantation. Patients (N = 58) received CCyd for up to 9 28-day cycl...

2014
Artur Jurczyszyn Wojciech Legieć Grzegorz Helbig Marek Hus Sławomira Kyrcz-Krzemień Aleksander B. Skotnicki

Carfilzomib (CFZ), an epoxyketone with specific chymotrypsin-like activity, is a second-generation proteasome inhibitor with significant activity in patients with relapsed and refractory multiple myeloma. On July 20, 2012, the US Food and Drug Administration approved CFZ to treat patients with multiple myeloma who have received at least two prior therapies including bortezomib (BORT) and an imm...

Journal: :Molecular cancer therapeutics 2013
Davide Zecchin Valentina Boscaro Enzo Medico Ludovic Barault Miriam Martini Sabrina Arena Carlotta Cancelliere Alice Bartolini Emily H Crowley Alberto Bardelli Margherita Gallicchio Federica Di Nicolantonio

A critical step toward defining tailored therapy in patients with cancer is the identification of genetic interactions that may impair-or boost-the efficacy of selected therapeutic approaches. Cell models able to recapitulate combinations of genetic aberrations are important to find drug-genotype interactions poorly affected by the heterogeneous genetics of human tumors. In order to identify no...

2017
Irene Riz Robert G. Hawley

Tight junction protein 1 (TJP1) has recently been proposed as a biomarker to identify multiple myeloma (MM) patients most likely to respond to bortezomib- and carfilzomib-based proteasome inhibitor regimens. Herein we report increased expression of TJP1 during the adaptive response mediating carfilzomib resistance in the LP-1/Cfz MM cell line. Moreover, increased TJP1 expression delineated a su...

2013
Joel G. Turner Jana Dawson Michael F. Emmons Christopher L. Cubitt Michael Kauffman Sharon Shacham Lori A. Hazlehurst Daniel M. Sullivan

Multiple myeloma (MM) remains an incurable disease despite improved treatments, including lenalidomide/pomalidomide and bortezomib/carfilzomib based therapies and high-dose chemotherapy with autologous stem cell rescue. New drug targets are needed to further improve treatment outcomes. Nuclear export of macromolecules is misregulated in many cancers, including in hematological malignancies such...

Journal: :Molecular and cellular endocrinology 2018
Claudia Busonero Stefano Leone Cinzia Klemm Filippo Acconcia

Most cases of breast cancer (BC) are estrogen receptor α-positive (ERα+) at diagnosis. The presence of ERα drives the therapeutic approach for this disease, which often consists of endocrine therapy (ET). 4OH-Tamoxifen and faslodex (i.e., fulvestrant - ICI182,780) are two ETs that render tumor cells insensitive to 17β-estradiol (E2)-dependent proliferative stimuli and prevent BC progression. Ho...

Journal: :Blood 2017
Changchun Deng Mark R Lipstein Luigi Scotto Xavier O Jirau Serrano Michael A Mangone Shirong Li Jeremie Vendome Yun Hao Xiaoming Xu Shi-Xian Deng Ronald B Realubit Nicholas P Tatonetti Charles Karan Suzanne Lentzsch David A Fruman Barry Honig Donald W Landry Owen A O'Connor

Phosphoinositide 3-kinase (PI3K) and the proteasome pathway are both involved in activating the mechanistic target of rapamycin (mTOR). Because mTOR signaling is required for initiation of messenger RNA translation, we hypothesized that cotargeting the PI3K and proteasome pathways might synergistically inhibit translation of c-Myc. We found that a novel PI3K δ isoform inhibitor TGR-1202, but no...

2013
Davide Zecchin Valentina Boscaro Enzo Medico Ludovic Barault Miriam Martini Sabrina Arena Carlotta Cancelliere Alice Bartolini Emily H. Crowley Alberto Bardelli Margherita Gallicchio Federica Di Nicolantonio

A critical step toward defining tailored therapy in patients with cancer is the identification of genetic interactions that may impair—or boost—the efficacy of selected therapeutic approaches. Cell models able to recapitulate combinations of genetic aberrations are important to find drug–genotype interactions poorly affected by the heterogeneous genetics of human tumors. In order to identify no...

Journal: :European journal of haematology 2011
Maurizio Zangari Monette Aujay Fenghuang Zhan Kristina L Hetherington Tamara Berno Ravi Vij Sundar Jagannath David Siegel A Keith Stewart Luhua Wang Robert Z Orlowski Andrew Belch Andrzej Jakubowiak George Somlo Suzanne Trudel Nizar Bahlis Sagar Lonial Seema Singhal Vishal Kukreti Guido Tricot

The ubiquitin-proteasome pathway regulates bone formation through osteoblast differentiation. We analyzed variation alkaline phosphatase (ALP) during carfilzomib treatment. Data from 38 patients enrolled in the PX-171-003 and 29 patients in PX-171-004 studies, for patients with relapsed/refractory myeloma, were analyzed. All patients received 20 mg/m(2) of carfilzomib on Days 1, 2, 8, 9, 15, an...

2017
Salam Kadhem Rawaa Ebrahem Scott Cooper Emily Manlove Ricky Lee

Posterior reversible encephalopathy syndrome (PRES) is a neurological condition characterized by headaches, visual disturbances, and seizures. A magnetic resonance imaging (MRI) scan of an affected brain typically shows symmetrical white matter edema in the posterior cerebral hemispheres. The onset of PRES can constitute a medical emergency, especially when accompanied by status epilepticus. If...

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