نتایج جستجو برای: developmental hepatotoxicity

تعداد نتایج: 129894  

Journal: :JAMA 2000
M S Sulkowski D L Thomas R E Chaisson R D Moore

CONTEXT Use of antiretroviral drugs, including protease inhibitors, for treatment of human immunodeficiency virus (HIV) infection has been anecdotally associated with hepatotoxicity, particularly in persons coinfected with hepatitis C or B virus. OBJECTIVES To ascertain if incidence of severe hepatotoxicity during antiretroviral therapy is similar for all antiretroviral drug combinations, and...

Journal: :Seminars in Liver Disease 2019

Journal: :Gastroenterology 1974

Journal: :Arthritis & Rheumatism 1977

Journal: :Oncology reports 2012
Hiroki Nishikawa Mitsuru Tsudo Yukio Osaki

Clinical outcome of diffuse large B-cell lymphoma (DLBCL) patients with hepatitis C virus (HCV) infection in the rituximab era remains unclear. The aim of the present study was to compare the clinical outcome, treatment response and hepatotoxicity in DLBCL patients who received rituximab containing immunochemotherapy that had HCV infection and tho...

Journal: :Yakugaku zasshi : Journal of the Pharmaceutical Society of Japan 2013
Yasuhiro Ishihara

Quinones are widely distributed in nature, and some quinone compounds are used as therapeutic agents such as anti-cancer, anti-malarial or anti-bacterial drugs. However, their therapeutic use is limited in some cases because the use of most quinones is accompanied by adverse effects derived from their cytotoxicity, especially for hepatocytes. Two mechanisms have been proposed to explain quinone...

2016
Oguzhan Sitki Dizdar Alparslan Ersoy Savas Aksoy Banu Demet Ozel Coskun Abdulmecit Yildiz

OBJECTIVE Immunosuppressive drugs, antimicrobial agents and infectious complications may cause liver function test abnormalities (LFTA) in kidney transplant recipients (KTR). The objectives of this study were to identify the outcome of (LFTA). To identify the risk factors affecting development and severity of hepatotoxicity in KTR. METHODS We retrospectively evaluated the medical records of K...

Journal: :Drug metabolism and disposition: the biological fate of chemicals 2007
Kristina K Wolf Sheryl G Wood Jenna L Allard Jane A Hunt Nadia Gorman Brooke W Walton-Strong Juliana G Szakacs Su X Duan Qin Hao Michael H Court Lisa L von Moltke David J Greenblatt Vsevolod Kostrubsky Elizabeth H Jeffery Steven A Wrighton Frank J Gonzalez Peter R Sinclair Jacqueline F Sinclair

CYP2E1 is widely accepted as the sole form of cytochrome P450 responsible for alcohol-mediated increases in acetaminophen (APAP) hepatotoxicity. However, we previously found that alcohol [ethanol and isopentanol (EIP)] causes increases in APAP hepatotoxicity in Cyp2e1(-/-) mice, indicating that CYP2E1 is not essential. Here, using wild-type and Cyp2e1(-/-) mice, we investigated the relative rol...

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