A Correction to this paper has been published: https://doi.org/10.1038/s41556-021-00638-5.

This paper aims to give a brief about the services provided by libraries disable library users using software and hardware technologies. In this era, development of technologies, information communication etc., is widespread being disseminated published globally. Usually, it convenient for professional differentiate between individuals in need assistance. emphasises specialised equipment, that ...

In this paper, a system for monitoring client-side activity is described. The system is capable of monitoring a wide range of user actions performed in web browser environment. Event logs are sent to the web server for further processing. The described system is completely script-driven on the client side, requiring no additional software to be installed on the client system and guaranteeing pl...

We advance first-mover advantage (FMA) theory by invoking two environmental dynamics – the pace of market evolution and the pace of technology evolution. Integrating several streams of literature, we elaborate on the interplay between these dynamics and the “isolating mechanisms” that underpin FMA. We draw a model of specific environmental conditions that potentially enable or disable FMA, and ...

Mobile computer users often have a false sense of anonymity when they connect to the Internet at cafes, hotels, airports or other public places. In this paper, we analyze information leaked by mobile computers to the local access link when they are outside their home domain. While most application data can be encrypted, there is no similar protection for signaling messages in the lower layers o...

Herpes Simplex Virus type 1 (HSV-1) has evolved to disable the cellular DNA damage response kinase, ATR. We have previously shown that HSV-1-infected cells are unable to phosphorylate the ATR substrate Chk1, even under conditions in which replication forks are stalled. Here we report that the HSV-1 single stranded DNA binding protein (ICP8), and the helicase/primase complex (UL8/UL5/UL52) form ...