نتایج جستجو برای: mdm2 snp309

تعداد نتایج: 4579  

2012
Simona Ognjanovic Ghyslaine Martel Carlos Manivel Magali Olivier Erica Langer Pierre Hainaut

The tumor suppressor gene TP53 is the most commonly mutated gene in human cancer. The reported prevalence of mutations in rhabdomyosarcoma (RMS) varies widely, with recent larger studies suggesting that TP53 mutations in pediatric RMS may be extremely rare. Overexpression of MDM2 also attenuates p53 function. We have performed TP53 mutation/MDM2 amplification analyses in the largest series anal...

2013
Ying-Yu Ma Tian-Pei Guan Hai-Bo Yao Sheng Yu Le-Gao Chen Ying-Jie Xia Xu-Jun He Hui-Ju Wang Xiao-Ting Jiang Hou-Quan Tao

BACKGROUND Recently, there have been a number of studies on the association between MDM2 (Murine Double Minute 2) 309 polymorphism and ovarian cancer risk. However, the results of previous reports remain controversial and ambiguous. Thus, we performed a meta-analysis to explore more precisely the association between MDM2 309 polymorphism and the risk of ovarian cancer. METHODS A meta-analysis...

Journal: :Asian Pacific journal of cancer prevention : APJCP 2015
Taweesak Tongtawee Chavaboon Dechsukhum Krajang Talabnin Wilairat Leeanansaksiri Soraya Kaewpitoon Natthawut Kaewpitoon Ryan A Loyd Likit Matrakool Sukij Panpimanmas

BACKGROUND The commonly held view of the tumor suppressor p53 is as a regulator of cell proliferation, apoptosis and many other biological processes as well as external and internal stress responses. Mdm2 SNIP309 is a negative regulator of p 53. Therefore, this study aimed to determine the correlation between the patterns of Mdm2 SNIP 309 and the inflammation grading of Helicobacter pylori asso...

Journal: :Blood 2008
Nathan A Ellis Dezheng Huo Ozlem Yildiz Lisa J Worrillow Mekhala Banerjee Michelle M Le Beau Richard A Larson James M Allan Kenan Onel

The p53 tumor suppressor directs the cellular response to many mechanistically distinct DNA-damaging agents and is selected against during the pathogenesis of therapy-related acute myeloid leukemia (t-AML). We hypothesized that constitutional genetic variation in the p53 pathway would affect t-AML risk. Therefore, we tested associations between patients with t-AML (n = 171) and 2 common functio...

Journal: :Cancer Epidemiology Biomarkers & Prevention 2008

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