نتایج جستجو برای: neurometabolic disorder

تعداد نتایج: 597203  

Journal: :Frontiers in Pharmacology 2021

The neurobiological bases of mood instability are poorly understood. Neuronal network alterations and neurometabolic abnormalities have been implicated in the pathophysiology anxiety conditions associated with hence candidate mechanisms underlying its neurobiology. Fast-spiking parvalbumin GABAergic interneurons modulate activity principal excitatory neurons through their inhibitory action dete...

Journal: :NMR in biomedicine 2010
M Henneke S Dreha-Kulaczewski K Brockmann M van der Graaf M A A P Willemsen U Engelke P Dechent A Heerschap G Helms R A Wevers J Gärtner

Adenylosuccinate lyase (ADSL) deficiency is an inherited metabolic disorder affecting predominantly the central nervous system. The disease is characterized by the accumulation of succinylaminoimidazolecarboxamide riboside and succinyladenosine (S-Ado) in tissue and body fluids. Three children presented with muscular hypotonia, psychomotor delay, behavioral abnormalities, and white matter chang...

2014
William J Zinnanti Jelena Lazovic Cathy Housman David A Antonetti David M Koeller James R Connor Lawrence Steinman

BACKGROUND Metabolic stroke is the rapid onset of lasting central neurological deficit associated with decompensation of an underlying metabolic disorder. Glutaric aciduria type I (GA1) is an inherited disorder of lysine and tryptophan metabolism presenting with metabolic stroke in infancy. The clinical presentation includes bilateral striatal necrosis and spontaneous subdural and retinal hemor...

2015
Dominique Endres Evgeniy Perlov Simon Maier Bernd Feige Kathrin Nickel Peter Goll Emanuel Bubl Thomas Lange Volkmar Glauche Erika Graf Dieter Ebert Esther Sobanski Alexandra Philipsen Ludger Tebartz van Elst

Attention-deficit hyperactivity disorder (ADHD) is a common neurodevelopmental disorder. In an attempt to extend earlier neurochemical findings, we organized a magnetic resonance spectroscopy (MRS) study as part of a large, government-funded, prospective, randomized, multicenter clinical trial comparing the effectiveness of specific psychotherapy with counseling and stimulant treatment with pla...

2010
Joanna D. Stewart Rita Horvath Enrico Baruffini Iliana Ferrero Stefanie Bulst Paul B. Watkins Robert J. Fontana Christopher P. Day Patrick F. Chinnery

Sodium valproate (VPA) is widely used throughout the world to treat epilepsy, migraine, chronic headache, bipolar disorder, and as adjuvant chemotherapy. VPA toxicity is an uncommon but potentially fatal cause of idiosyncratic liver injury. Rare mutations in POLG, which codes for the mitochondrial DNA polymerase c (polc), cause Alpers-Huttenlocher syndrome (AHS). AHS is a neurometabolic disorde...

2012
Barbara Cellini Riccardo Montioli Elisa Oppici Carla Borri Voltattorni

Dopa decarboxylase (DDC) is a pyridoxal 5'-phosphate (PLP)-dependent enzyme that by catalyzing the decarboxylation of L-Dopa and L-5-hydroxytryptophan produces the neurotransmitters dopamine and serotonin. The functional properties of pig kidney and human DDC enzymes have been extensively characterized, and the crystal structure of the enzyme in the holo- and apo-forms has been elucidated. DDC ...

2011
Maria Puiu Cristina Rusu Corin Badiu Anca Botezatu Natalia Cucu

Prader Willi Syndrome (PWS) is a neurometabolic genetic disorder affecting 1/12.000-1/15.000 newborns. Molecular mechanisms that could lead to this disorder include chromosomal deletions, uniparental disomy (UPD), intragenic mutations, and epigenetic modifications in the process of imprinting and rarely reciprocal translocations. A common defect is noticed in all cases: loss of parental contrib...

Journal: :Brain : a journal of neurology 2010
Michèl A Willemsen Marcel M Verbeek Erik-Jan Kamsteeg Johanneke F de Rijk-van Andel Alec Aeby Nenad Blau Alberto Burlina Maria A Donati Ben Geurtz Padraic J Grattan-Smith Martin Haeussler Georg F Hoffmann Hans Jung Johannis B de Klerk Marjo S van der Knaap Fernando Kok Vincenzo Leuzzi Pascale de Lonlay Andre Megarbane Hugh Monaghan Willy O Renier Pierre Rondot Monique M Ryan Jürgen Seeger Jan A Smeitink Gerry C Steenbergen-Spanjers Evangeline Wassmer Bernhard Weschke Frits A Wijburg Bridget Wilcken Dimitrios I Zafeiriou Ron A Wevers

Tyrosine hydroxylase deficiency is an autosomal recessive disorder resulting from cerebral catecholamine deficiency. Tyrosine hydroxylase deficiency has been reported in fewer than 40 patients worldwide. To recapitulate all available evidence on clinical phenotypes and rational diagnostic and therapeutic approaches for this devastating, but treatable, neurometabolic disorder, we studied 36 pati...

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