نتایج جستجو برای: nitric oxide synthase 3

تعداد نتایج: 1999609  

Journal: :Circulation 2012
Manling Zhang Eiki Takimoto Dong-ik Lee Celio X C Santos Taishi Nakamura Steven Hsu Aiyang Jiang Takahiro Nagayama Djahida Bedja Yuan Yuan Philip Eaton Ajay M Shah David A Kass

BACKGROUND In the normal heart, phosphodiesterase type 5 (PDE5) hydrolyzes cGMP coupled to nitric oxide- (specifically from nitric oxide synthase 3) but not natriuretic peptide (NP)-stimulated guanylyl cyclase. PDE5 is upregulated in hypertrophied and failing hearts and is thought to contribute to their pathophysiology. Because nitric oxide signaling declines whereas NP-derived cGMP rises in su...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2010
Fiona D Saunders Martin Westphal Perenlei Enkhbaatar Jianpu Wang Konrad Pazdrak Yoshimitsu Nakano Atsumori Hamahata Collette C Jonkam Matthias Lange Rhykka L Connelly Gabriela A Kulp Robert A Cox Hal K Hawkins Frank C Schmalstieg Eszter Horvath Csaba Szabo Lillian D Traber Elbert Whorton David N Herndon Daniel L Traber

Neuronal nitric oxide synthase is critically involved in the pathogenesis of acute lung injury resulting from combined burn and smoke inhalation injury. We hypothesized that 7-nitroindazole, a selective neuronal nitric oxide synthase inhibitor, blocks central molecular mechanisms involved in the pathophysiology of this double-hit insult. Twenty-five adult ewes were surgically prepared and rando...

Journal: :The New England journal of medicine 2005
Mark J D Griffiths Timothy W Evans

Nitric oxide was largely regarded as a toxic pollutant until 1987, when its biologic similarities to endothelium-derived relaxing factor were demonstrated.1 Subsequently, nitric oxide and endothelium-derived relaxing factor were considered a single entity, modulating vascular tone through the stimulated formation of cyclic guanosine 3',5'-monophosphate (Fig. 1).2 Endogenous nitric oxide is form...

Background: Liver ischemia / reperfusion Injury (IRI) is one of the major causes of liver failure during various types of liver surgery, trauma and infections. The present study investigates the effect of dexsamethasone on the liver injury and inducible nitric oxide synthase gene expression during hepatic warm ischemia/reperfusion in rats. Materials and Methods: 24 male Wistar rats (200-250 g)...

2000
Daniel A. Kahn D. Clay Archer Carolyn J. Kelly

Journal: :The Journal of urology 1997
M A Wheeler S D Smith N Saito H E Foster R M Weiss

PURPOSE We attempted to determine whether oral L-arginine, the substrate for nitric oxide synthase, increases nitric oxide synthase activity and cyclic guanosine monophosphate (cGMP) levels in the urine from interstitial cystitis patients. Nitric oxide and cGMP are decreased in urine from interstitial cystitis patients and both induce smooth muscle relaxation and immunological responses. Increa...

Journal: :Experimental and clinical transplantation : official journal of the Middle East Society for Organ Transplantation 2013
Negar Azarpira Mahdokht H Aghdai Bita Geramizadeh Ali Bahador Maryam Ayatolahi Masumeh Darai

OBJECTIVES Nitric oxide is a major mediator in vascular biology and regulator of regional blood flow. Its production is catalyzed by the enzyme endothelial nitric oxide synthase. Protective actions of nitric oxide in ischemia and reperfusion are due to its potential as an antioxidant and anti-inflammatory agent, along with its inhibitory effects on cell signaling pathways of nuclear proteins, s...

Journal: :Hypertension research in pregnancy 2023

Aim: This study aimed to investigate the effects of relaxin (RLX) on hypertensive disorders pregnancy (HDP) and fetal growth restriction (FGR) using a nitric oxide synthase (NOS) inhibitor-induced HDP murine model.

Journal: :Pharmacology, biochemistry, and behavior 2015
Semil Selcen Gocmez Yusufhan Yazir Deniz Sahin Sabriye Karadenizli Tijen Utkan

Since the discovery of nitric oxide (NO) as a neuronal messenger, its way to modulate learning and memory functions is subject of intense research. NO is an intercellular messenger in the central nervous system and is formed on demand through the conversion of L-arginine to L-citrulline via the enzyme nitric oxide synthase (NOS). Neuronal form of nitric oxide synthase may play an important role...

Journal: :Hypertension 2009
Victor V Lima Fernanda R C Giachini Hyehun Choi Fernando S Carneiro Zidonia N Carneiro Zuleica B Fortes Maria Helena C Carvalho R Clinton Webb Rita C Tostes

Hyperglycemia, which increases O-linked beta-N-acetylglucosamine (O-GlcNAc) proteins, leads to changes in vascular reactivity. Because vascular dysfunction is a key feature of arterial hypertension, we hypothesized that vessels from deoxycorticosterone acetate and salt (DOCA-salt) rats exhibit increased O-GlcNAc proteins, which is associated with increased reactivity to constrictor stimuli. Aor...

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