نتایج جستجو برای: neuronal apoptosis inhibitory protein (naip) gene.

تعداد نتایج: 2222941  

Journal: :medical journal of islamic republic of iran 0
seyed reza kazemi nezhad department of genetics, faculty of science, shahid chamran universityof ahvaz, ahvaz, iran.سازمان اصلی تایید شده: دانشگاه شهید چمران (shahid chamran university) fatemeh mosavi department of genetics, faculty of science, shahid chamran university of ahvaz, ahvaz, iran.سازمان اصلی تایید شده: دانشگاه شهید چمران (shahid chamran university) ali akbar momen ahvaz jundishapur university of medical sciences, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی جندی شاپور اهواز (ahvaz jundishapur university of medical sciences) hamid galehdari department of genetics, faculty of science, shahid chamran university of ahvaz, ahvaz, iran.سازمان اصلی تایید شده: دانشگاه شهید چمران (shahid chamran university) gholamreza mohamadian genetic counseling centre, khuzestan welfare organization, ahvaz, iran.سازمان های دیگر: khuzestan welfare organization

background: spinal muscular atrophy (sma) is the second most common lethal autosomal recessive disease. it is a neuromuscular disorder caused by degenerative of lower motor neurons and occasionally bulbar neurons leading to progressive limb paralysis and muscular atrophy. the smn1 gene is recognized as a sma causing gene while naip has been characterized as a modifying factor for the clinical s...

Journal: :iranian journal of child neurology 0
mohammad barzegar professor of pediatric neurology, pediatric health research center, tabriz university of medical sciences, tabriz, iran maryam shoaran pediatrician, faculty of medicine, tabriz university of medical sciences, children hospital, tabriz, iran mortaza bonyadi associate professor of molecular-medical genetics, faculty of natural sciences. tabriz university, tabriz, iran

objective we describe three patients with very severe spinal muscular atrophy (sma) presented with reduced fetal movement in utero, profound hypotonia, severe weakness and respiratory insufficiency at birth. in all infants, electrodiagnostic studies were compatible with a neurogenic pattern. in genetic studies, all cases had homozygous deletions of exons 7 and 8 of survival motor neuron (smn) a...

Journal: :American journal of physiology. Renal physiology 2006
Alison Dziarmaga Pierre-Alain Hueber Diana Iglesias Nancy Hache Aaron Jeffs Nathalie Gendron Alex Mackenzie Michael Eccles Paul Goodyer

During fetal kidney development, the extent of ureteric bud (UB) branching will determine final nephron endowment for life. Nephron number varies widely among normal humans and those who are born at the low end of the nephron number spectrum may be at risk for essential hypertension in adulthood. Little is known about how nephron number is set. However, we previously showed that the transcripti...

2006
Alison Dziarmaga Pierre-Alain Hueber Diana Iglesias Nancy Hache Aaron Jeffs Nathalie Gendron Alex MacKenzie Michael Eccles Paul Goodyer

During fetal kidney development, the extent of ureteric bud (UB) branching will determine final nephron endowment for life. Nephron number varies widely among normal humans and those who are born at the low end of the nephron number spectrum may be at risk for essential hypertension in adulthood. Little is known about how nephron number is set. However, we have previously shown that the transcr...

Journal: :genetics in the 3rd millennium 0
امید آریانی omid ariani مرتضی بنیادی morteza bonyadi center of excellence for molecular analysis of smn gene biodiversity, department of genetics, faculty of natural sciences, university of tabriz, tabriz, iran محمد برزگر mohammad barzegar

spinal muscular atrophy (sma) is an autosomal recessive neuromuscular disorder characterized by degeneration of spinal cord anterior horn cells, leading to muscular atrophy. sma is clinically classified into three sub-groups based on the age of onset and severity. the majority of patients with sma have homozygous deletions of exons 7 and 8 of the survival motor neuron (smn) gene. the purpose of ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2002
Johannes K X Maier Zahia Lahoua Nathalie H Gendron Raouf Fetni Anne Johnston Jamshid Davoodi Dita Rasper Sophie Roy Ruth S Slack Donald W Nicholson Alex E MacKenzie

The neuronal apoptosis inhibitory protein (NAIP) was identified as a candidate gene for the inherited neurodegenerative disorder spinal muscular atrophy. NAIP is the founding member of a human protein family that is characterized by highly conserved N-terminal motifs called baculovirus inhibitor of apoptosis repeats (BIR). Five members of the human family of inhibitor of apoptosis proteins incl...

Journal: :Cell 1995
Natalie Roy Mani S Mahadevan Michael McLean Gary Shutter Zahra Yaraghi Reza Farahani Stephen Baird Anne Besner-Johnston Charles Lefebvre Xiaolin Kang Maysoon Salih Huguette Aubry Katsuyuki Tamai Xiaoping Guan Panayiotis Ioannou Thomas O Crawford Pieter J de Jong Linda Surh Joh-E Ikeda Robert G Korneluk Alex MacKenzie

The spinal muscular atrophies (SMAs), characterized by spinal cord motor neuron depletion, are among the most common autosomal recessive disorders. One model of SMA pathogenesis invokes an inappropriate persistence of normally occurring motor neuron apoptosis. Consistent with this hypothesis, the novel gene for neuronal apoptosis inhibitory protein (NAIP) has been mapped to the SMA region of ch...

Journal: :PLoS ONE 2009
Mark T. Romanish Hisae Nakamura C. Benjamin Lai Yuzhuo Wang Dixie L. Mager

The human neuronal apoptosis inhibitory protein (NAIP) gene is no longer principally considered a member of the Inhibitor of Apoptosis Protein (IAP) family, as its domain structure and functions in innate immunity also warrant inclusion in the Nod-Like Receptor (NLR) superfamily. NAIP is located in a region of copy number variation, with one full length and four partly deleted copies in the ref...

Journal: :Human molecular genetics 2000
R Götz C Karch M R Digby J Troppmair U R Rapp M Sendtner

The human neuronal apoptosis inhibitory protein (NAIP) gene has been discovered as a candidate gene for spinal muscular atrophy, a genetic disorder characterized by motor neuron loss in the spinal cord. The telomeric NAIP gene on human chromosome 5 is deleted together with survival motor neurons (SMN) in many cases of the most severe forms of the disorder. NAIP, c-IAP1 (inhibitor of apoptosis-1...

Journal: :Cancer letters 2010
Helen H L Chiu Theresa M K Yong Jun Wang Yuwei Wang Robert L Vessella Takeshi Ueda Yu-Zhuo Wang Marianne D Sadar

A mechanism for survival of prostate cancer cells in an androgen-deprived environment remains elusive. Here, we find that expression of neuronal apoptosis inhibitory protein (NAIP) was significantly increased in vivo and in vitro in response to androgen deprivation therapy (ADT). Increased expression of NAIP corresponded to increased DNA-binding activity of NF-kappaB that physically associated ...

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