A unified model for the molecular basis of Xeroderma pigmentosum-Cockayne Syndrome

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A unified model for the molecular basis of Xeroderma pigmentosum-Cockayne Syndrome

Nucleotide Excision Repair (NER) is a pathway that removes lesions distorting the DNA helix. The molecular basis of the rare diseases Xeroderma pigmentosum (XP) and Cockayne Syndrome (CS) are explained based on the defects happening in 2 NER branches: Global-Genome Repair and Transcription-Coupled Repair, respectively. Nevertheless, both afflictions sporadically occur together, giving rise to X...

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Xeroderma pigmentosum-Cockayne syndrome complex

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Xeroderma pigmentosum--Cockayne syndrome complex: a further case.

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Rare case of Cockayne syndrome with xeroderma pigmentosum.

Sir, Xeroderma pigmentosum (XP) and Cockayne syndrome (CS) are rare genodermatoses characterized by hypersensitivity to ultraviolet light. XP is a hereditary autosomal recessive condition, manifested clinically by abnormal photosensitivity of the skin and eyes, pigment anomalies, increased risk of cutaneous neoplasms and, frequently, by progressive neural degeneration (1 ± 3). CS is a rare auto...

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Reduced RNA polymerase II transcription in extracts of cockayne syndrome and xeroderma pigmentosum/Cockayne syndrome cells.

The hereditary disease Cockayne syndrome (CS) is a complex clinical syndrome characterized by arrested post-natal growth as well as neurological and other defects. The CSA and CSB genes are implicated in this disease. The clinical features of CS can also accompany the excision repair-defective hereditary disorder xeroderma pigmentosum (XP) from genetic complementation groups B, D or G. The XPB ...

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ژورنال

عنوان ژورنال: Rare Diseases

سال: 2015

ISSN: 2167-5511

DOI: 10.1080/21675511.2015.1079362