Endothelial Precursor Cell Migration During Vasculogenesis
نویسندگان
چکیده
منابع مشابه
Mechanisms of Vascular Smooth Muscle Cell Migration Endothelial Cell Migration During Angiogenesis Endothelial Precursor Cell Migration During Vasculogenesis Endothelial-Dependent Mechanisms of Leukocyte Recruitment to the Vascular Wall Molecular Mechanisms of Endothelial Cell Migration
Inflammation is a fundamental process that protects organisms by removing or neutralizing injurious agents. A key event in the inflammatory response is the localized recruitment of various leukocyte subsets. Here we address the cellular and regulatory mechanisms of leukocyte recruitment to the vessel wall in cardiovascular disease and discuss our evolving understanding of the role of the vascul...
متن کاملMechanisms of Vascular Smooth Muscle Cell Migration Endothelial Cell Migration During Angiogenesis Endothelial Precursor Cell Migration During Vasculogenesis Leukocyte Migration in the Vascular Wall Molecular Mechanisms of Endothelial Cell Migration
Migration of endothelial precursor cells (so-called “angioblasts” in embryos and “endothelial progenitor cells” in adults) during vasculogenesis is a requirement for the formation of a primary vascular plexus. The migration is initiated by the change of endothelial precursors to their migratory phenotype. The endothelial precursor cells are then guided to the position where the primary vascular...
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Vasculogenesis and angiogenesis are critical processes in fetal circulation and placental vasculature development. Placental mesenchymal stem cells (pMSC) are known to release paracrine factors (some of which are contained within exosomes) that promote angiogenesis and cell migration. The aims of this study were: to determine the effects of oxygen tension on the release of exosomes from pMSC; a...
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A dietary deficiency of vitamin A is associated with cardiovascular abnormalities in avian and murine systems. Retinoic acid (RA) is the active metabolite of vitamin A and whether it directly regulates mammalian blood vessel formation has not been determined and is investigated herein. We used mice rendered RA-deficient via targeted deletion of retinaldehyde dehydrogenase 2 (Raldh2(-/-)), the e...
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ژورنال
عنوان ژورنال: Circulation Research
سال: 2007
ISSN: 0009-7330,1524-4571
DOI: 10.1161/circresaha.107.148932