Maximum Aerobic Exercise Induces Common Carotid Vasoconstriction And Increased Vascular Resistance

Michael Diaz, Zaria Opara, Heysel Morales, Steven Hawkins, Louise Kelly, Allan Knox PURPOSE: Low-to-moderate intensity aerobic exercise has been reported to elicit changes in systemic hemodynamics. Recent evidence suggests that the cerebral circulation reacts independently circulations same physiological stimuli. The aim of this is determine influence high-intensity on common carotid artery (CCA) METHODS: Hemodynamic measurements were recorded before and following cessation a maximum oxygen uptake test (VO2max). One minute video CCA by Doppler ultrasound used peak systolic velocity (PSV), end diastolic (EDV), time averaged mean blood (TAMEAN), timed average (TAMAX), pulsatility index (PI), resistance (RI). diameters calculated commercially available vessel tracking software. Carotid stiffness (β), Peterson’s pressure modulus (Ep), arterial compliance (AC) was also calculated. RESULTS: A total 29 individuals, 14 male 15 females, participated study (21.9 ± 3.9 years). Following exercise, PSV (p < 0.001), TAMAX = 0.0085), RI PI 0.0032) significantly increased whilst minimum diameter 0.005), 0.020) decreased. No observed for β 0.491), Ep 0.834) or AC 0.535). CONCLUSION: results show hemodynamic VO2max exercise. Specifically, increases vascular are likely due reductions diameter, which may be evident downstream CCA. Acute maximal does not change parameters.