Reactive Oxygen Species: Integral Components of B Cell Activation

Mitochondrial Reactive Oxygen Species Mediate Lysophosphatidylcholine-Induced Endothelial Cell Activation.

OBJECTIVE Hyperlipidemia-induced endothelial cell (EC) activation is considered as an initial event responsible for monocyte recruitment in atherogenesis. However, it remains poorly defined what is the mechanism underlying hyperlipidemia-induced EC activation. Here, we tested a novel hypothesis that mitochondrial reactive oxygen species (mtROS) serve as signaling mediators for EC activation in ...

Stimulation Promotes B Cell Activation and Species in Response to B Cell Receptor Prolonged Production of Reactive Oxygen

Proliferation Stimulation Promotes B Cell Activation and Species in Response to B Cell Receptor Prolonged Production of Reactive Oxygen

Update on Reactive Oxygen Species in Plant Cell Death Reactive Oxygen Species in Plant Cell Death

Paradoxically, death is an integral part of life. Cell death is essential for growth and development of eukaryotes, by maintaining tissue and organ homeostasis in concert with cell proliferation, growth, and differentiation. Until recently, the wide variety of cell death types reported in the literature was mostly caged in two semantic categories: apoptosis and necrosis. Discrimination between ...

O 22: Reactive Oxygen Species and Epilepsy

Seizure activity has been proposed to result in the generation of reactive oxygen species (ROS), which then contribute to seizure-induced neuronal damage and eventually cell death. Although the mechanisms of seizure-induced ROS generation are unclear, mitochondria and cellular calcium overload have been proposed to have a crucial role. We aim to determine the sources of seizure-induced ROS and ...