the changing epidemiology of herpes simplex virus type 1 infection: the associated effects on the incidence of ocular herpes

b. abedi kiasari; department of human viral vaccines, razi vaccine and serum research institute, karaj, iran z. zare tooranposhti; department of clinical and molecular virology, kiatech virology laboratory, karaj, iran

مقاله

the changing epidemiology of herpes simplex virus type 1 infection: the associated effects on the incidence of ocular herpes

b. abedi kiasari, department of human viral vaccines, razi vaccine and serum research institute, karaj, iran z. zare tooranposhti, department of clinical and molecular virology, kiatech virology laboratory, karaj, iran

چکیده(به انگلیسی؛ چکیده‌ی فارسی در دسترس نیست)

herpes simplex virus type 1 (hsv-1) with a worldwide distribution has been reported in all human populations, resulting in a clinical spectrum of infections. although hsv type 2 (hsv-2) is known as the most common cause of genital herpes, an increasing number of cases with genital herpes are caused by hsv-1. the present study aimed to discuss the changes in the epidemiology of hsv-1 infection including the decline in the general incidence of hsv-1 infection in childhood and the increased rate of genital herpes, caused by hsv-1. moreover, changes in the epidemiology of ocular herpes, i.e., the reduced rate of primary ocular herpes in children and increased incidence of ocular hsv infection in adults, were discussed.

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b. abedi kiasari, department of human viral vaccines, razi vaccine and serum research institute, karaj, iran z. zare tooranposhti, department of clinical and molecular virology, kiatech virology laboratory, karaj, iran (n.d.). the changing epidemiology of herpes simplex virus type 1 infection: the associated effects on the incidence of ocular herpes. archives of razi institute. https://www.virascience.com/article/115462/

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  title = {the changing epidemiology of herpes simplex virus type 1 infection: the associated effects on the incidence of ocular herpes},
  author = {b. abedi kiasari and department of human viral vaccines, razi vaccine and serum research institute, karaj, iran z. zare tooranposhti and department of clinical and molecular virology, kiatech virology laboratory, karaj, iran},
  journal = {archives of razi institute},
  url = {https://www.virascience.com/article/115462/}
}

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کلیدواژه‌ها

اسناد مشابه

A 437-base-pair deletion at the beginning of the latency-associated transcript promoter significantly reduced adrenergically induced herpes simplex virus type 1 ocular reactivation in latently infected rabbits.
سند علمی۱۹۹۷Journal of virologyJ M Hill, H H Garza, Y H Su, R Meegalla
In this study we used a herpes simplex virus type 1 (HSV-1) deletion mutant to identify a segment of the genome necessary for epinephrine-induced reactivation in the rabbit eye model of herpetic recurrent disease. In HSV-1 latently infected neural tissue, the only abundant viral
Multiplex polymerase chain reaction for detection of herpes simplex virus type 1, type 2, cytomegalovirus, and varicella-zoster virus in ocular viral infections.
سند علمی۲۰۰۳Japanese journal of ophthalmologyYan Zhang, Tairo Kimura, Keiko Fujiki, Hitoshi Sakuma
PURPOSE To detect simultaneously herpes simplex virus type 1 (HSV-1), type 2 (HSV-2), cytomegalovirus (CMV), and varicella-zoster virus (VZV) in ocular specimens suspected of indicating viral infection, and to compare the results of multiplex polymerase chain reaction (PCR) with
Th-cytotoxic T-lymphocyte chimeric epitopes extended by Nepsilon-palmitoyl lysines induce herpes simplex virus type 1-specific effector CD8+ Tc1 responses and protect against ocular infection.
سند علمی۲۰۰۵Journal of virologyXiuli Zhang, Annie Issagholian, Eric A Berg, Jordan B Fishman
Molecularly defined vaccine formulations capable of inducing antiviral CD8+ T-cell-specific immunity in a manner compatible with human delivery are limited. Few molecules achieve this target without the support of an appropriate immunological adjuvant. In this study, we investiga
Abnormal immune response of CCR5-deficient mice to ocular infection with herpes simplex virus type 1.
سند علمی۲۰۰۶The Journal of general virologyDaniel J J Carr, John Ash, Thomas E Lane, William A Kuziel
Ocular herpes simplex virus type 1 (HSV-1) infection elicits a strong inflammatory response that is associated with production of the beta chemokines CCL3 and CCL5, which share a common receptor, CCR5. To gain insight into the role of these molecules in ocular immune responses, t
Protection against recurrent ocular herpes simplex virus type 1 disease after therapeutic vaccination of latently infected mice.
سند علمی۲۰۰۳Journal of virologyC M Richards, R Case, T R Hirst, T J Hill
The potential of therapeutic vaccination of animals latently infected with herpes simplex virus type 1 (HSV-1) to enhance protective immunity to the virus and thereby reduce the incidence and severity of recurrent ocular disease was assessed in a mouse model. Mice latently infect
Efficacy of a recombinant glycoprotein D subunit vaccine on the development of primary and recurrent ocular infection with herpes simplex virus type 1 in mice.
سند علمی۱۹۹۷The Journal of infectious diseasesT L Keadle, K A Laycock, J K Miller, K K Hook
The protective efficacy of a glycoprotein D subunit vaccine (gD2 SB AS4) was evaluated in a mouse model of human recurrent herpetic stromal keratitis (HSK). When administered before primary infection, gD2 SB AS4 protected mice against corneal pathology, mortality, and latency res

the changing epidemiology of herpes simplex virus type 1 infection: the associated effects on the incidence of ocular herpes

چکیده(به انگلیسی؛ چکیده‌ی فارسی در دسترس نیست)

اسناد مشابه

A 437-base-pair deletion at the beginning of the latency-associated transcript promoter significantly reduced adrenergically induced herpes simplex virus type 1 ocular reactivation in latently infected rabbits.

Multiplex polymerase chain reaction for detection of herpes simplex virus type 1, type 2, cytomegalovirus, and varicella-zoster virus in ocular viral infections.

Th-cytotoxic T-lymphocyte chimeric epitopes extended by Nepsilon-palmitoyl lysines induce herpes simplex virus type 1-specific effector CD8+ Tc1 responses and protect against ocular infection.

Abnormal immune response of CCR5-deficient mice to ocular infection with herpes simplex virus type 1.

Protection against recurrent ocular herpes simplex virus type 1 disease after therapeutic vaccination of latently infected mice.

Efficacy of a recombinant glycoprotein D subunit vaccine on the development of primary and recurrent ocular infection with herpes simplex virus type 1 in mice.