Mdm2 associates with Ras effector NORE1 to induce the degradation of oncoprotein HIPK1.

نویسندگان

  • Deresa Lee
  • Sang-Joon Park
  • Ki Sa Sung
  • Jikyoung Park
  • Sean Bong Lee
  • Sang-Yoon Park
  • Hyo Jeong Lee
  • Jang-Won Ahn
  • So Jung Choi
  • Seok-Geun Lee
  • Sung-Hoon Kim
  • Duk-Hwan Kim
  • Jhingook Kim
  • Yongsok Kim
  • Cheol Yong Choi
چکیده

The Ras effector NORE1 is frequently silenced in primary adenocarcinomas, although the significance of this silencing for tumorigenesis is unclear. Here we show that NORE1 induces polyubiquitination and proteasomal degradation of oncoprotein HIPK1 by facilitating its interaction with the Mdm2 E3 ubiquitin ligase. Endogenous HIPK1 is stabilized in Nore1-deficient mouse embryonic fibroblasts, and depletion of HIPK1 in NORE1-silenced lung adenocarcinoma cells inhibits anchorage-independent cell growth and tumour formation in nude mice. These findings indicate that the control of HIPK1 stability by Mdm2-NORE1 has a major effect on cell behaviour, and epigenetic inactivation of NORE1 enables adenocarcinoma formation in vivo through HIPK1 stabilization.

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عنوان ژورنال:
  • EMBO reports

دوره 13 2  شماره 

صفحات  -

تاریخ انتشار 2012