A positive feedback loop between mesenchymal-like cancer cells and macrophages is essential to breast cancer metastasis.

نویسندگان

  • Shicheng Su
  • Qiang Liu
  • Jingqi Chen
  • Jianing Chen
  • Fei Chen
  • Chonghua He
  • Di Huang
  • Wei Wu
  • Ling Lin
  • Wei Huang
  • Jin Zhang
  • Xiuying Cui
  • Fang Zheng
  • Haiyan Li
  • Herui Yao
  • Fengxi Su
  • Erwei Song
چکیده

The close vicinity of cancer cells undergoing epithelial-mesenchymal transition (EMT) and tumor-associated macrophages (TAMs) at the invasive front of tumors suggests that these two cell type may mutually interact. We show that mesenchymal-like breast cancer cells activate macrophages to a TAM-like phenotype by GM-CSF. Reciprocally, CCL18 from TAMs induces cancer cell EMT, forming a positive feedback loop, in coculture systems and humanized mice. Inhibition of GM-CSF or CCL18 breaks this loop and reduces cancer metastasis. High GM-CSF expression in breast cancer samples is associated with more CCL18(+) macrophages, cancer cell EMT, enhanced metastasis, and reduced patient survival. These findings suggest that a positive feedback loop between GM-CSF and CCL18 is important in breast cancer metastasis.

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عنوان ژورنال:
  • Cancer cell

دوره 25 5  شماره 

صفحات  -

تاریخ انتشار 2014