نتایج جستجو برای: ethylmalonic aciduria

تعداد نتایج: 1386  

2013
Mathilde Nizon Chris Ottolenghi Vassili Valayannopoulos Jean-Baptiste Arnoux Valérie Barbier Florence Habarou Isabelle Desguerre Nathalie Boddaert Jean-Paul Bonnefont Cécile Acquaviva Jean-François Benoist Daniel Rabier Guy Touati Pascale de Lonlay

BACKGROUND Classical organic acidurias including methylmalonic aciduria (MMA), propionic aciduria (PA) and isovaleric aciduria (IVA) are severe inborn errors of the catabolism of branched-chain amino acids and odd-numbered chain fatty acids, presenting with severe complications. METHODS This study investigated the long-term outcome of 80 patients with classical organic aciduria (38 with MMA, ...

Journal: :Anais da Academia Brasileira de Ciencias 2015
Nádia W Dimer Patrícia F Schuck Emilio L Streck Gustavo C Ferreira

Inherited metabolic diseases are a heterogeneous group of diseases caused by a punctual defect in cell metabolism, resulting in the accumulation of toxic intermediate metabolites or in the lack of important biomolecules for adequate cell functioning. D-glyceric aciduria is an inherited disease caused by a deficiency of glycerate 2-kinase activity, whose pathophysiological mechanisms remain unkn...

Journal: :Blood 1977
E P Frenkel R L Kitchens

A rapid sensitive spectrophotometric assay for the measurement of methylmalonic and propionic acids in urine is described. The assay is based upon the quantitation of propionic acid using acetyl coenzyme A synthetase isolated from baker's yeast. This enzyme is highly specific for acetate and propionate, and acetate interference is eliminated by conversion to citrate. Methylmalonic acid was assa...

Journal: :The Journal of biological chemistry 2003
Heidi Peters Mikhail Nefedov Joseph Sarsero James Pitt Kerry J Fowler Sophie Gazeas Stephen G Kahler Panayiotis A Ioannou

Methylmalonic aciduria is a human autosomal recessive disorder of organic acid metabolism resulting from a functional defect in the activity of the enzyme methylmalonyl-CoA mutase. Based upon the homology of the human mutase locus with the mouse locus, we have chosen to disrupt the mouse mutase locus within the critical CoA binding domain using gene-targeting techniques to create a mouse model ...

Journal: :Journal of Pediatric Neurosciences 2009

Journal: :Archives of disease in childhood 1983
C R Roe C L Hoppel T E Stacey R A Chalmers B M Tracey D S Millington

Patients with methylmalonic aciduria have an excessive intramitochondrial accumulation of acylcoenzyme A compounds that may reduce the availability of free coenzyme A (CoA) for normal metabolic requirements, producing profound metabolic disturbances. Giving carnitine to a patient with methylmalonic aciduria produced an increase in hippurate excretion (an index of intramitochondrial adenosine tr...

Journal: :BMC Pediatrics 2002
Fernando Scaglia Angela E Scheuerle Jeffrey A Towbin Dawna L Armstrong Lawrence Sweetman Lee-Jun C Wong

BACKGROUND Hyperammonemia, hypoglycemia, hepatopathy, and ventricular tachycardia are common presenting features of carnitine-acylcarnitine translocase deficiency (Mendelian Inheritance in Man database: *212138), a mitochondrial fatty acid oxidation disorder with a lethal prognosis. These features have not been identified as the presenting features of mitochondrial cytopathy in the neonatal per...

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