نتایج جستجو برای: ethylmalonic aciduria

تعداد نتایج: 1386  

2017
Marina A Morath

Ethylmalonic encephalopathy is a devastating, infantile, autosomal recessive, metabolic disorder caused by defects in the mitochondrial sulfur dioxygenase, ETHE1, and characterized by ethylmalonic and methylsuccinic aciduria, lactic acidemia associated with developmental delay, orthostatic acrocyanosis, recurrent petechiae, chronic diarrhea, and abnormalities on brain MRI. The authors also repo...

Journal: :Archives of neurology 2004
Karen A McGowan William L Nyhan Bruce A Barshop Robert K Naviaux Alice Yu Richard H Haas Jeannette J Townsend

BACKGROUND Among patients with ethylmalonic aciduria, a subgroup with encephalopathy, petechial skin lesions, and often death in infancy is distinct from those with short-chain acyl-coenzyme A dehydrogenase deficiency or multiple acyl-coenzyme A dehydrogenase deficiency. The nature of the molecular defect in this subgroup is unknown, and the source of the ethylmalonic acid has been unclear. O...

Journal: :Journal of Clinical Investigation 1979

Journal: :iranian journal of child neurology 0
alireza tavasoli 1. pediatric neurology division, neurometabolic registry center, children’s medical center, tehran university of medical sciences, tehran, iran parastoo rostami 2. division of endocrinology and metabolism, department of pediatrics, children’s medical center, tehran university of medical sciences, tehran, iran mahmoud reza ashrafi 1. pediatric neurology division, neurometabolic registry center, children’s medical center, tehran university of medical sciences, tehran, iran parvaneh karimzadeh 3. pediatric neurology, pediatric neurology research center, shahid beheshti university of medical sciences, tehran, iran 4.pediatric neurology excellence center, pediatric neurology department, mofid children hospital, faculty of medicine, shahid beheshti university of medical sciences (sbmu), tehran, iran

how to cite this article: tavasoli ar, rostami p, ashrafi mr, karimzadeh p. neurological and vascular manifestations of ethylmalonic encephalopathy. iran j child neurol. spring 2017; 11(2):57-60.   abstract objective ethylmalonic encephalopathy (ee) is a severe mitochondrial disease of early infancy clinically characterized by a combination of developmental delay, progressive pyramidal signs, a...

Journal: :iranian journal of child neurology 0
parvaneh karimzadeh associate professor of pediatric neurology, pediatric neurology research center, shahidbeheshti university of medical sciences, mofid children hospital, tehran, iran

this condition is due to deficiency in electron transport flavoprotein or electron transport fluvoprotein dehydrogenase.the clinical presentation is characteristic of fatty acid oxidation disorders. this disorder is poor prognostic and death in infancy is common.the central nervous system involving causes neurodevelopmental delay, hypotonia, and head lag. different type of seizures such as infa...

Journal: :Scholars Journal of Applied Medical Sciences 2020

Journal: :Human molecular genetics 1998
N Gregersen V S Winter M J Corydon T J Corydon P Rinaldo A Ribes G Martinez M J Bennett C Vianey-Saban A Bhala D E Hale W Lehnert S Kmoch M Roig E Riudor H Eiberg B S Andresen P Bross L A Bolund S Kølvraa

We have shown previously that a variant allele of the short-chain acyl-CoA dehydrogenase ( SCAD ) gene, 625G-->A, is present in homozygous form in 7% of control individuals and in 60% of 135 patients with elevated urinary excretion of ethylmalonic acid (EMA). We have now characterized three disease-causing mutations (confirmed by lack of enzyme activity after expression in COS-7 cells) and a ne...

Journal: :The Journal of clinical investigation 1979
S Mantagos M Genel K Tanaka

The mechanisms underlying ethylmalonic-adipic aciduria were studied in a 5-yr-old girl. Oxidation of radioactive substrates by cultured skin fibroblasts from the proband and asymptomatic family members was also determined and compared to that by normal fibroblasts and that by cells from a patient with glutaric aciduria type II. Feeding medium-chain triglycerides promptly induced vomiting and le...

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