نتایج جستجو برای: vwf

تعداد نتایج: 3173  

ژورنال: :فصلنامه پژوهشی خون 0
محمدرحیم فاتحی فر m.r. fatehifar مرکز تحقیقات انتقال خون ـ مؤسسه عالی آموزشی و پژوهشی طب انتقال خون و پایگاه منطقهای آموزشی انتقال خون اصفهان حمید هورفر h. horfar دانشگاه علوم پزشکی اصفهانسازمان اصلی تایید شده: سازمان انتقال خون ایران (blood transfusion research center) ناهید اکبری n. akbari مؤسسه عالی آموزشی و پژوهشی طب انتقال خون و پایگاه منطقهای آموزشی انتقال خونسازمان اصلی تایید شده: دانشگاه علوم پزشکی اصفهان (isfahan university of medical sciences) محسن اشراقی m eshraghi کارشناس هنر ـ رئیس کانون هموفیلی ایران نمایندگی استان اصفهان مهدی حجتی فر m. hojatifar کارشناس علوم آزمایشگاهی ـ بیمارستان سیدالشهدا)ع( اصفهان ـ اصفهان نصرت معتمدی n. motamedi کاردان پرستاری ـ بیمارستان سیدالشهدا)ع( اصفهان زهرا شاهین پور

چکید ه   سابقه و هدف   تمایل به خونریزی در ناقلین هموفیلی a از سال ها قبل شناخته شده به طوری که برخی حتی با سطح فاکتور 40% تا 60% نیز، دچار خونریزی های غیر طبیعی می شوند. هدف از مطالعه تعیین سطح فاکتور viii و فون ویلبراند و بررسی ارتباط آن ها با علایم بالینی در ناقلین قطعی بود.   مواد و روش ها   این بررسی توصیفی بر روی 49 زن ناقل قطعی هموفیلی a در سال 1389 انجام شد. آزمایش ptt ، pt ، f. viii و ...

Journal: :Heart 2003
D A Gorog H Douglas N Ahmed D C Lefroy G J Davies

Shear induced platelet activation is responsible for arterial thrombosis. The role of fibrinogen is established at low shear stress, but at high shear stress von Willebrand factor (vWF), not fibrinogen, is essential for platelet adhesion and aggregation. Patients with vWF deficiency exhibit resistance to thrombosis, and raised vWF is a risk factor for acute coronary syndromes. Interpretation of...

Journal: :Blood 2000
S Allen A M Abuzenadah J Hinks J L Blagg T Gursel J Ingerslev A C Goodeve I R Peake M E Daly

In this report we describe the molecular defect underlying partial and severe quantitative von Willebrand factor (VWF) deficiencies in 3 families previously diagnosed with types 1 and 3 Von Willebrand-disease. Analysis of the VWF gene in affected family members revealed a novel C to T transition at nucleotide 1067 of the VWF complemetary DNA (cDNA), predicting substitution of arginine by trypto...

Journal: :Thrombosis and haemostasis 2014
Volker Huck Matthias F Schneider Christian Gorzelanny Stefan W Schneider

The specific interactions of von Willebrand factor (VWF) with the vessel wall, platelets or other interfaces strongly depend on (a shear-induced) VWF activation. Shear flow has been shown to induce a conformational transition of VWF, but is modulated by its thermodynamic state (state-function relationship). The state in turn is determined by physical (e.g. vessel geometry), physico-chemical (e....

Journal: :Blood cells, molecules & diseases 2010
Maira Carrillo Seongcheol Kim Surendra Kumar Rajpurohit Vrinda Kulkarni Pudur Jagadeeswaran

von Willebrand factor (vWF) is a large protein involved in primary hemostasis. A dysfunction in this protein or an insufficient production of the protein leads to improper platelet adhesion/aggregation, resulting in a bleeding phenotype known as von Willebrand disease (vWD). To gain a better understanding of vWF interactions in vivo, the use of zebrafish as a model is ideal because of the trans...

2006
Ronan Pendu Virginie Terraube Olivier D. Christophe Carl G. Gahmberg Philip G. de Groot Peter J. Lenting Cecile V. Denis

Von Willebrand factor (VWF) is an essential component of hemostasis. However, animal studies using VWF-deficient mice suggest that VWF may also contribute to inflammation. In the present study, we demonstrate that VWF was able to interact with polymorphonuclear leukocytes (PMNs) and monocytes under static and flow conditions. Adhesion under flow was dominated by short-lasting contact with resti...

Journal: :Blood 2014
Luiza Rusu Alexandra Andreeva David J Visintine Kyungho Kim Stephen M Vogel Aleksandra Stojanovic-Terpo Olga Chernaya Guoquan Liu Farnaz R Bakhshi Sandra L Haberichter Hiroko Iwanari Osamu Kusano-Arai Nobuchika Suzuki Takao Hamakubo Tohru Kozasa Jaehyung Cho Xiaoping Du Richard D Minshall

von Willebrand factor (vWF) secretion by endothelial cells (ECs) is essential for hemostasis and thrombosis; however, the molecular mechanisms are poorly understood. Interestingly, we observed increased bleeding in EC-Gα13(-/-);Gα12(-/-) mice that could be normalized by infusion of human vWF. Blood from Gα12(-/-) mice exhibited significantly reduced vWF levels but normal vWF multimers and impai...

Journal: :Blood 2008
Anil K Chauhan Meghan T Walsh Guojing Zhu David Ginsburg Denisa D Wagner David G Motto

Ultralarge von Willebrand factor (UL-VWF) multimers are thought to play a central role in pathogenesis of the disease thrombotic thrombocytopenic purpura (TTP); however, experimental evidence in support of this hypothesis has been difficult to establish. Therefore, to examine directly the requirement for VWF in TTP pathogenesis, we generated ADAMTS13-deficient mice on a TTP-susceptible genetic ...

Journal: :Haematologica 2009
Almudena Pérez-Rodríguez Aranzazu García-Rivero Esther Lourés Maria Fernanda López-Fernández Angela Rodríguez-Trillo Javier Batlle

BACKGROUND Mutation C1149R in the von Willebrand factor (VWF) gene has been thought to cause autosomal dominant severe type 1 von Willebrand disease (VWD). DESIGN AND METHODS Eight patients from three unrelated families with this mutation were included in the present study who had distinct VWF abnormalities, not described in earlier studies. RESULTS The patients showed notably low levels of...

Journal: :Nucleic acids research 1985
C. L. Verweij C. J. de Vries B. Distel A. J. van Zonneveld A. Geurts van Kessel J. A. van Mourik H. Pannekoek

Von Willebrand Factor (vWF) mRNA was identified in fractionated polyA+ RNA preparations isolated from cultured human endothelial cells. Micro-injection of specific polyA+ RNA fractions in Xenopus laevis oocytes provoked the synthesis of a vWF-like product which could be detected with an immunoradiometric assay relying on Sepharose-linked monoclonal anti-vWF IgG and different radiolabeled monocl...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید