نتایج جستجو برای: hypertrophic atrial fibrillation

تعداد نتایج: 121477  

Journal: :Stroke 2008
Jacob I Haft

Echocardiographic Findings That May Help Identify Occult Intermittent Atrial Fibrillation in Hypertensive Patients at Risk for a Second (or First) Ischemic Stroke To the Editor: In their review, Liao et al1 found that Holter or event monitoring of patients after ischemic stroke identified 5% to 7% with intermittent atrial fibrillation not present on the admission ECG. They suggest that such mon...

A Pakizeh Moghadam, J Haddadnia, M Eskandari M J Monaghan

Background: Mechanical occlusion of the Left atrial appendage (LAA) using a purpose-built device has emerged as an effective prophylactic treatment in patients with atrial fibrillation at risk of stroke and a contraindication for anticoagulation. A crucial step in procedural planning is the choice of the device size. This is currently based on the manual analysis of the “Device Landing Zone” fr...

Journal: :Circulation 1966
M H Luria E I Adelson A J Miller

THE INTRODUCTION of the adrenergic beta-receptor blocking agent, pronethalol,1 and the more recent availability of its analogue propranolol2 have served as a new impetus in the pharmacological study of the cardiovascular system. Clinical use of these compounds has been explored in the treatment of hypertension,3 angina pectoris,4 prolonged systemic hypotension,5 hypertrophic subaortic stenosis,...

Introduction: Electrophysiological studies have demonstrated a relationship between aging and atrioventricular (AV) nodal conduction and refractoriness. The aim of the present study was to determine the effects of nodal aging on dynamic AV nodal field potential recording during atrial fibrillation (AF) in rabbit. Methods: Two groups of male New Zealand rabbits (neonatal 2-week-olds and adult...

2013
Christina Westphal Bastian Spallek Anne Konkel Lajos Marko Fatimunnisa Qadri Laura M. DeGraff Carola Schubert J. Alyce Bradbury Vera Regitz-Zagrosek John R. Falck Darryl C. Zeldin Dominik N. Müller Wolf-Hagen Schunck Robert Fischer

Maladaptive cardiac hypertrophy predisposes one to arrhythmia and sudden death. Cytochrome P450 (CYP)-derived epoxyeicosatrienoic acids (EETs) promote anti-inflammatory and antiapoptotic mechanisms, and are involved in the regulation of cardiac Ca(2+)-, K(+)- and Na(+)-channels. To test the hypothesis that enhanced cardiac EET biosynthesis counteracts hypertrophy-induced electrical remodeling, ...

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