نتایج جستجو برای: caspase programmed cell death

تعداد نتایج: 1883179  

Journal: :The Journal of comparative neurology 2000
Q Zeng A Kwan B Oakley

In the adult mouse tongue, an average of 11% of the gustatory receptor cells are replaced each day. In investigating homeostatic cell death mechanisms in gustatory renewing epithelium, we observed that taste receptor cells were selectively immunopositive for the bcl-2 family death factor, Bax, and for the protease Caspase-2 (Nedd2/Ich1). We determined that 8-10% of the taste receptor cells of t...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2017
Lisa P Daley-Bauer Linda Roback Lynsey N Crosby A Louise McCormick Yanjun Feng William J Kaiser Edward S Mocarski

The complex interplay between caspase-8 and receptor-interacting protein (RIP) kinase RIP 3 (RIPK3) driving extrinsic apoptosis and necroptosis is not fully understood. Murine cytomegalovirus triggers both apoptosis and necroptosis in infected cells; however, encoded inhibitors of caspase-8 activity (M36) and RIP3 signaling (M45) suppress these antiviral responses. Here, we report that this vir...

2014
Paramita Banerjee Sawant Aritra Bera Subrata Dasgupta Bhawesh T. Sawant Narinder K. Chadha Asim K. Pal

Hypoxia is a global phenomenon affecting recruitment as well as the embryonic development of aquatic fauna. The present study depicts hypoxia induced disruption of the intrinsic pathway of programmed cell death (PCD), leading to embryonic malformation in the goldfish, Carrasius auratus. Constant hypoxia induced the early expression of pro-apoptotic/tumor suppressor p53 and concomitant expressio...

2012
Marie Anne O’Donnell Hidenori Hase Diana Legarda Adrian T. Ting

TNF can trigger two opposing responses: cell survival and cell death. TNFR1 activates caspases that orchestrate apoptosis but some cell types switch to a necrotic death when treated with caspase inhibitors. Several genes that are required to orchestrate cell death by programmed necrosis have been identified, such as the kinase RIP1, but very little is known about the inhibitory signals that kee...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Dan Weng Robyn Marty-Roix Sandhya Ganesan Megan K Proulx Gregory I Vladimer William J Kaiser Edward S Mocarski Kimberly Pouliot Francis Ka-Ming Chan Michelle A Kelliher Phillip A Harris John Bertin Peter J Gough Dmitry M Shayakhmetov Jon D Goguen Katherine A Fitzgerald Neal Silverman Egil Lien

A number of pathogens cause host cell death upon infection, and Yersinia pestis, infamous for its role in large pandemics such as the "Black Death" in medieval Europe, induces considerable cytotoxicity. The rapid killing of macrophages induced by Y. pestis, dependent upon type III secretion system effector Yersinia outer protein J (YopJ), is minimally affected by the absence of caspase-1, caspa...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید