نتایج جستجو برای: hepatic c

تعداد نتایج: 1139720  

Journal: :Computational and Mathematical Methods in Medicine 2011

Journal: :Transplantation 1990
T Ikeda K Yanaga G Lebeau H Higashi S Kakizoe T E Starzl

Using an ex vivo liver sanguinous perfusion system, hemodynamic and biochemical changes of the porcine livers were studied, which were preserved cold (4 degrees C) for 24 hr in University of Wisconsin solution and reperfused with normothermic (37 degrees C) (n = 8) or hypothermic (32 degrees C) (n = 8) blood for 3 hr. Six more livers were reperfused with normothermic blood (37 degrees C) immedi...

2014
Minoru Tomizawa Fuminobu Shinozaki Yasufumi Motoyoshi Takao Sugiyama Shigenori Yamamoto

Liver recapitulates the same size as before partial hepatectomy (PH). Once hepatectomized, tumor necrosis factor (TNF) a and interleukin (IL)-6 are secreted from non-parenchymal cells. The cytokines promotes hepatocyte proliferation via signal transducer and activator of transcription 3. Hepatocyte growth factor and epidermal growth factor are potent mitogen of hepatocytes. CCAAT/enhancer bindi...

Journal: :Cancer 2003
Kazuyuki Ohata Keisuke Hamasaki Kan Toriyama Kojiro Matsumoto Akira Saeki Kenji Yanagi Seigou Abiru Yuichi Nakagawa Masaya Shigeno Seiji Miyazoe Tatsuki Ichikawa Hiroki Ishikawa Kazuhiko Nakao Katsumi Eguchi

BACKGROUND Hepatic steatosis is one of the histopathologic features of chronic hepatitis C. It was reported recently that the expression of hepatitis C virus (HCV) core protein in transgenic mice induced hepatocellular carcinoma (HCC) in association with steatosis. The objective of this study was to determine the relation between hepatic steatosis and hepatocarcinogenesis in patients with chron...

2017
John P. Gibbs J. Greg Slatter Ogo Egbuna Michelle Geller Lisa Hamilton Clapton S. Dias Ren Y. Xu Jessica Johnson Scott M. Wasserman Maurice G. Emery

Evolocumab binds PCSK9, increasing low-density lipoprotein cholesterol (LDL-C) receptors and lowering LDL-C. Target-mediated evolocumab elimination is attributable to PCSK9 binding. As circulating PCSK9 and LDL-C levels are primarily regulated by the liver, we compared evolocumab pharmacokinetics, pharmacodynamics, and safety in individuals with and without hepatic impairment. An open-label, pa...

Journal: :Diabetes 2003
Aruna Asipu Bruce E Hayward John O'Reilly David T Bonthron

Alternative splicing of the ketohexokinase (fructokinase) gene generates a "central" predominantly hepatic isoform (ketohexokinase-C) and a more widely distributed ketohexokinase-A. Only the abundant hepatic isoform is known to possess activity, and no function is defined for the lower levels of ketohexokinase-A in peripheral tissues. Hepatic ketohexokinase deficiency causes the benign disorder...

Journal: :Journal of Gastroenterology and Hepatology 2013

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