نتایج جستجو برای: locomotor deficits

تعداد نتایج: 65456  

Journal: :Human Molecular Genetics 2009
Micaela Galante Harsha Jani Lesley Vanes Hervé Daniel Elizabeth M.C. Fisher Victor L.J. Tybulewicz Timothy V.P. Bliss Elise Morice

Down syndrome (DS) is a genetic disorder arising from the presence of a third copy of human chromosome 21 (Hsa21). Recently, O'Doherty et al. [An aneuploid mouse strain carrying human chromosome 21 with Down syndrome phenotypes. Science 309 (2005) 2033-2037] generated a trans-species aneuploid mouse line (Tc1) that carries an almost complete Hsa21. The Tc1 mouse is the most complete animal mode...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2008
John P Vessey Paolo Macchi Joel M Stein Martin Mikl Kelvin N Hawker Petra Vogelsang Krzysztof Wieczorek Georgia Vendra Julia Riefler Fabian Tübing Samuel A J Aparicio Ted Abel Michael A Kiebler

The dsRNA-binding protein Staufen was the first RNA-binding protein proven to play a role in RNA localization in Drosophila. A mammalian homolog, Staufen1 (Stau1), has been implicated in dendritic RNA localization in neurons, translational control, and mRNA decay. However, the precise mechanisms by which it fulfills these specific roles are only partially understood. To determine its physiologi...

2011
Yousuf O. Ali Wilfredo Escala Kai Ruan R. Grace Zhai

Advances in genetic methods have enabled the study of genes involved in human neurodegenerative diseases using Drosophila as a model system. Most of these diseases, including Alzheimer's, Parkinson's and Huntington's disease are characterized by age-dependent deterioration in learning and memory functions and movement coordination. Here we use behavioral assays, including the negative geotaxis ...

2016
Martin Weber Tiffany Wu Jesse E. Hanson Nazia M. Alam Hilda Solanoy Hai Ngu Benjamin E. Lauffer Han H. Lin Sara L. Dominguez Jens Reeder Jennifer Tom Pascal Steiner Oded Foreman Glen T. Prusky Kimberly Scearce-Levie

Age is the main risk factor for sporadic Alzheimer’s disease. Yet, cognitive decline in aged rodents has been less well studied, possibly due to concomitant changes in sensory or locomotor function that can complicate cognitive tests. We tested mice that were 3, 11, and 23 months old in cognitive, sensory, and motor measures, and postmortem measures of gliosis and neural activity (c-Fos). Hippo...

Journal: :Brain, behavior, and immunity 2009
Linnéa Asp Simret Beraki Krister Kristensson Sven Ove Ogren Håkan Karlsson

Epidemiological studies suggest that early life infections may contribute to the development of psychiatric disorders characterized by cognitive deficits. Here, we studied the effects of a neonatal influenza A/WSN/33 virus infection on locomotor activity, working memory and emotional behavior in adult mice. In addition to wild type mice, immunodeficient (Tap1(-/-)) mice lacking functional CD8(+...

2015
Joshua T. Dearborn Steven K. Harmon Stephen C. Fowler Karen L. O’Malley George T. Taylor Mark S. Sands David F. Wozniak

Infantile neuronal ceroid lipofuscinosis (INCL, Infantile Batten disease) is a neurodegenerative lysosomal storage disease caused by a deficiency in palmitoyl protein thioesterase-1 (PPT1). The PPT1-deficient mouse (Cln1(-/-)) is a useful phenocopy of human INCL. Cln1(-/-) mice display retinal dysfunction, seizures, motor deficits, and die at ~8 months of age. However, little is known about the...

2011
Julien Vezoli Karim Fifel Vincent Leviel Colette Dehay Henry Kennedy Howard M. Cooper Claude Gronfier Emmanuel Procyk

BACKGROUND It is increasingly recognized that non-motor symptoms are a prominent feature of Parkinson's disease and in the case of cognitive deficits can precede onset of the characteristic motor symptoms. Here, we examine in 4 monkeys chronically treated with low doses of the neurotoxin MPTP the early and long-term alterations of rest-activity rhythms in relationship to the appearance of motor...

Journal: :Behavioural brain research 1997
S Maren G Aharonov M S Fanselow

Electrolytic lesions of the dorsal hippocampus (DH) produce deficits in both the acquisition and expression of conditional fear to contextual stimuli in rats. To assess whether damage to DH neurons is responsible for these deficits, we performed three experiments to examine the effects of neurotoxic N-methyl-D-aspartate (NMDA) lesions of the DH on the acquisition and expression of fear conditio...

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