نتایج جستجو برای: oncogenic viruse

تعداد نتایج: 21746  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2003
Alan Charest Vicky Kheifets Julie Park Keara Lane Kevin McMahon Cathy L Nutt David Housman

Activating oncogenic mutations of receptor tyrosine kinases (RTKs) have been reported in several types of cancers. In many cases, genomic rearrangements lead to the fusion of unrelated genes to the DNA coding for the kinase domain of RTKs. All RTK-derived fusion proteins reported so far display oligomerization sequences within the 5' fusion partners that are responsible for oncogenic activation...

Journal: :Cancer research 2013
Yaqing Zhang Wei Yan Meredith A Collins Filip Bednar Sabita Rakshit Bruce R Zetter Ben Z Stanger Ivy Chung Andrew D Rhim Marina Pasca di Magliano

Pancreatic cancer, one of the deadliest human malignancies, is almost invariably associated with the presence of an oncogenic form of Kras. Mice expressing oncogenic Kras in the pancreas recapitulate the stepwise progression of the human disease. The inflammatory cytokine interleukin (IL)-6 is often expressed by multiple cell types within the tumor microenvironment. Here, we show that IL-6 is r...

2013
Masaki Takahashi Tomoko Chiyo Takashi Okada Hirohiko Hohjoh

Anticancer agents that have minimal effects on normal cells and tissues are ideal cancer drugs. Here, we show specific inhibition of human cancer cells carrying oncogenic mutations in the epidermal growth factor receptor (EGFR) gene by means of oncogenic allele-specific RNA interference (RNAi), both in vivo and in vitro. The allele-specific RNAi (ASP-RNAi) treatment did not affect normal cells ...

2017
Yaqing Zhang Wei Yan Esha Mathew Kevin T Kane Arthur Brannon Maeva Adoumie Alekya Vinta Howard C Crawford Marina Pasca di Magliano

Dedifferentiation of acini to duct-like cells occurs during the physiologic damage response in the pancreas, but this process can be co-opted by oncogenic Kras to drive carcinogenesis. Myeloid cells infiltrate the pancreas during the onset of pancreatic cancer, and promote carcinogenesis. Here, we show that the function of infiltrating myeloid cells is regulated by oncogenic Kras expressed in e...

Journal: :Cancer research 2015
Junko Tanizaki Dalia Ercan Marzia Capelletti Michael Dodge Chunxiao Xu Magda Bahcall Erin M Tricker Mohit Butaney Antonio Calles Lynette M Sholl Peter S Hammerman Geoffrey R Oxnard Kwok-Kin Wong Pasi A Jänne

The discovery of oncogenic driver mutations and the subsequent developments in targeted therapies have led to improved outcomes for subsets of lung cancer patients. The identification of additional oncogenic and drug-sensitive alterations may similarly lead to new therapeutic approaches for lung cancer. We identify and characterize novel FGFR2 extracellular domain insertion mutations and demons...

Journal: :Cancer research 1978
W F Benedict A Banerjee N Venkatesan

Cyclophosphamide, an extensively used cancer chemotherapeutic agent, requires metabolic activation through a mixed-function oxygenase system. The capacity of this agent to produce oncogenic transformation and chromosomal damage, including increases in sister chromatid exchanges, was investigated in cell culture with or without an exogenous liver metabolic activation system. No oncogenic transfo...

Journal: :Blood 2012
Ashley F Ward Benjamin S Braun Kevin M Shannon

Ras proteins are critical nodes in cellular signaling that integrate inputs from activated cell surface receptors and other stimuli to modulate cell fate through a complex network of effector pathways. Oncogenic RAS mutations are found in ∼25% of human cancers and are highly prevalent in hematopoietic malignancies. Because of their structural and biochemical properties, oncogenic Ras proteins a...

Journal: :Clinical and Experimental Otorhinolaryngology 2012

Journal: :Proceedings of the Royal Society of Medicine 1971

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