نتایج جستجو برای: family history fh

تعداد نتایج: 739379  

2017
Boyoung Park John L. Hopper Aung K. Win James G. Dowty Ho Kyung Sung Choonghyun Ahn Sung-Won Kim Min Hyuk Lee Jihyoun Lee Jong Won Lee Eunyoung Kang Jong-Han Yu Ku Sang Kim Byung-In Moon Wonshik Han Dong-Young Noh Sue K. Park

This study was conducted to identify the role of reproductive factors as environmental modifiers for breast cancer (BC) risk in clinic-based, East-Asian BRCA1 and BRCA2 mutation carriers and non-carriers with high-risk criteria of BRCA mutations (family history (FH) of BC, early-onset BC (aged ≤40 years)). A total of 581 women who were BRCA carriers (222 BRCA1 and 359 BRCA2), 1,083 non-carriers...

2015
Ranjit Manchanda Rosa Legood Matthew Burnell Alistair McGuire Maria Raikou Kelly Loggenberg Jane Wardle Saskia Sanderson Sue Gessler Lucy Side Nyala Balogun Rakshit Desai Ajith Kumar Huw Dorkins Yvonne Wallis Cyril Chapman Rohan Taylor Chris Jacobs Ian Tomlinson Uziel Beller Usha Menon Ian Jacobs

BACKGROUND Population-based testing for BRCA1/2 mutations detects the high proportion of carriers not identified by cancer family history (FH)-based testing. We compared the cost-effectiveness of population-based BRCA testing with the standard FH-based approach in Ashkenazi Jewish (AJ) women. METHODS A decision-analytic model was developed to compare lifetime costs and effects amongst AJ wome...

2015
Rena SHIGENAGA Sadako AKASHI-TANAKA Satoko UCHIDA Murasaki IKEDA Hiroto OYAMA Reiko YOSHIDA Kenya SUZUKI Katsutoshi ENOKIDO Terumasa SAWADA Junko YOTSUMOTO Seigo NAKAMURA

Germline mutations of BRCA1 / 2 genes cause hereditary breast and / or ovarian cancer. However, whether guidelines like those of the National Comprehensive Cancer Network (NCCN) can suitably predict the likelihood of BRCA1 / 2 mutations in the Japanese population is unclear. Methods BRCA1 / 2 gene mutation frequencies were investigated in relation to parameters such as age, family history (FH),...

2010
François Normand-Lauzière Frédérique Frisch Sébastien M. Labbé Patrick Bherer René Gagnon Stephen C. Cunnane André C. Carpentier

BACKGROUND It has been proposed that abnormal postprandial plasma nonesterified fatty acid (NEFA) metabolism may participate in the development of tissue lipotoxicity and type 2 diabetes (T2D). We previously found that non-diabetic offspring of two parents with T2D display increased plasma NEFA appearance and oxidation rates during intravenous administration of a fat emulsion. However, it is cu...

2004
George Miltiadous Marios A. Cariolou Moses Elisaf

Objective: To characterise the mutations in the LDL receptor gene in patients with heterozygous familial hypercholesterolaemia (FH) living in Northwestern Greece and to determine, if any, the geographical distribution of these mutations. Methods: DNA analysis for the LDL receptor gene was performed (using restriction enzyme method or direct sequencing) in unrelated patients attending our lipid ...

Journal: :Journal of immunology 2010
Isharat Yusuf Robin Kageyama Laurel Monticelli Robert J Johnston Daniel Ditoro Kyle Hansen Burton Barnett Shane Crotty

CD4 T cell help is critical for the generation and maintenance of germinal centers (GCs), and T follicular helper (T(FH)) cells are the CD4 T cell subset required for this process. Signaling lymphocytic activation molecule (SLAM)-associated protein (SAP [SH2D1A]) expression in CD4 T cells is essential for GC development. However, SAP-deficient mice have only a moderate defect in T(FH) different...

بنی طالبی, مهدی, شایسته , فاطمه, شیرانی, منوچهر, مبینی, غلامرضا, پروین, ندا , صفاری چالشتری, جواد , طاهرزاده فر رخشهری, مریم , قطره سامانی, کیهان , مدرسی, مهرداد , هاشم زاده, مرتضی,

چکیده: زمینه و هدف: کلسترول بالای خانوادگی (FH) یک اختلال اتوزوم غالب است که عمدتاً به علت جهش های ژن LDLR و ApoB-100ایجاد می شود. تاکنون اساس مولکولی FH بطور مفصل در بسیاری از جمعیت ها تشریح شده است ولی هنوز اطلاعات مولکولی اندکی در ارتباط با FH در ایران موجود است. هدف از این مطالعه بررسی فراوانی 3 جهش شایع ژنی آپولیپوپروتئین B-100 ((ApoB-100 در یک جمعیت ایرانی است. روش بررسی: در این مطالعه...

اسدی, سمیه, بنی طالبی, مهدی, حاجی حسینی بغداد آبادی, رضا, شایسته, فاطمه, صفاری چالشتری, جواد, طاهرزاده قهفرخی, مریم, قطره سامانی, کیهان, مبینی, غلامرضا, ناظم, حبیب اله, هاشم زاده چالشتری, مرتضی ,

چکیده: زمینه و هدف: هایپرکلسترولمی فامیلی (FH) بیماری غالب اتوزومال است که عمدتاً بدلیل جهش در ژن گیرنده لیپوپروتئین با دانسیته کم (LDLR) ایجاد می شود. این مطالعه با هدف بررسی تغییرات ژن LDLR بیماران مبتلا به کلسترول بالای خانوادگی در استان چهارمحال و بختیاری انجام شد. روش بررسی: در این مطالعه توصیفی- آزمایشگاهی، 57 بیمار مشکوک به FH غربالگری و با استفاده از روش PCR-SSCP جهش در پروموتر و اگز...

Journal: :Hypertension 2001
K Ishikawa S Baba T Katsuya N Iwai T Asai M Fukuda S Takiuchi Y Fu T Mannami J Ogata J Higaki T Ogihara

A common variant at codon 235 of the angiotensinogen gene with methionine to threonine amino acid substitution (AGT M235T) has been reported as a genetic risk for essential hypertension. However, the frequency of AGT T235 was heterogeneous among races, and a positive association between AGT M235T and hypertension was not settled. To examine the association in a general population of Japanese (n...

2017
Glenmore Lasam Siddesh Shambhu Robert Fishberg

We report a very young man with heterozygous familial hypercholesterolemia (FH) with APOE haplotype and a significant cardiac family history who underwent cardiac catheterization for intermittent episodes of exertional dyspnea and was noted to have a severe triple vessel coronary artery disease (CAD). He underwent coronary artery bypass graft (CABG) surgery which was uneventful. He was discharg...

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