نتایج جستجو برای: ras models

تعداد نتایج: 935979  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2009
Caroline Gregorian Jonathan Nakashima Sarah M Dry P Leia Nghiemphu Kate Barzan Smith Yan Ao Julie Dang Gregory Lawson Ingo K Mellinghoff Paul S Mischel Michael Phelps Luis F Parada Xin Liu Michael V Sofroniew Fritz C Eilber Hong Wu

Patients with neurofibromatosis type 1 (NF1) carry approximately a 10% lifetime risk of developing a malignant peripheral nerve sheath tumor (MPNST). Although the molecular mechanisms underlying NF1 to MPNST malignant transformation remain unclear, alterations of both the RAS/RAF/MAPK and PI3K/AKT/mTOR signaling pathways have been implicated. In a series of genetically engineered murine models,...

Journal: :Circulation 2007
Junwang Xu Fraz A Ismat Tao Wang Jifu Yang Jonathan A Epstein

BACKGROUND Neurofibromatosis type I (NF1) is a common autosomal dominant disorder with a broad array of clinical manifestations, including benign and malignant tumors, osseous dysplasias, and characteristic cutaneous findings. In addition, NF1 patients have an increased incidence of cardiovascular diseases, including obstructive vascular disorders. In animal models, endothelial expression of th...

Journal: :Cancer research 2003
Kumari L Andarawewa Anne Boulay Régis Masson Carole Mathelin Isabelle Stoll Catherine Tomasetto Marie-Pierre Chenard Martine Gintz Jean-Pierre Bellocq Marie-Christine Rio

In human carcinomas, stromelysin-3/matrix metalloproteinase 11 (ST3, MMP11) expression by nonmalignant fibroblastic cells located in the immediate vicinity of cancer cells is a bad prognostic factor. Using mouse models of primary tumors, it has been demonstrated that ST3 is a key player during local invasion, favoring cancer cell survival in connective tissue through an antiapoptotic function. ...

Journal: :Cancer research 2006
Yu-An Zhang John Nemunaitis Shirley K Samuel Patrick Chen Yuqiao Shen Alex W Tong

Despite successes in animal models, cancer gene therapy with small interfering RNAs (siRNA) is hindered by the lack of an optimal delivery platform. We examined the applicability of the replication-competent, oncolytic adenovirus, ONYX-411, to deliver a mutant K-ras siRNA transgene to human cancer cells. Proof-of-principle studies showed an additive tumor growth-inhibitory response through siRN...

Journal: :Journal of Flood Risk Management 2021

Digital surface models, land use and rainfall data were used to simulate water areas using Hydrologic Engineering Centres River Analysis System (HEC-RAS) software. Multi-temporal synthetic aperture radar (SAR) was for the detection of flood prone area calibrate HEC-RAS, due lack validation in New Cairo City, Egypt. The thresholding method applied two Sentinel-1 images, one pre- post-flash event...

Journal: :Frontiers in Immunology 2023

Introduction Tumor-associated macrophages may act to either limit or promote tumor growth, yet the molecular basis for path is poorly characterized. Methods We use a larval Drosophila model that expresses dominant-active version of Ras-oncogene (Ras V12 ) study dysplastic growth during early progression. performed single-cell RNA-sequencing macrophage-like hemocytes characterize these cells in ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2003
Steven S Clark Li Zhong Daniele Filiault Sarah Perman Zhibin Ren Michael Gould Xinhai Yang

PURPOSE Perillyl alcohol (POH) displays preventive and therapeutic activity against a wide variety of tumor models, and it has been suggested that this might be associated with the ability of POH to interfere with Ras prenylation. POH also selectively induces G(1) arrest and apoptosis in Bcr/Abl-transformed hematopoietic cells. Because signaling through Ras is necessary for Bcr/Abl transformati...

Journal: :Molecular cancer therapeutics 2002
Robert B Lobell Dongming Liu Carolyn A Buser Joseph P Davide Elizabeth DePuy Kelly Hamilton Kenneth S Koblan Yih Lee Scott Mosser Sherri L Motzel James L Abbruzzese Charles S Fuchs Eric K Rowinsky Eric H Rubin Sunil Sharma Paul J Deutsch Kathryn E Mazina Briggs W Morrison Lynne Wildonger Siu-Long Yao Nancy E Kohl

Farnesyl:protein transferase (FPTase) inhibitors were developed as anti-Ras drugs, but they fail to inhibit Ki-Ras activity because Ki-Ras can be modified by geranylgeranyl:protein transferase type-I (GGPTase-I). L-778,123, an inhibitor of FPTase and GGPTase-I, was developed in part because it can completely inhibit Ki-Ras prenylation. To support the clinical development of L-778,123, we develo...

Journal: :Molecular pharmacology 2010
Chun-Ki Kim Yoon Kyung Choi Hansoo Lee Kwon-Soo Ha Moo-Ho Won Young-Guen Kwon Young-Myeong Kim

Farnesyltransferase (FTase) inhibitors induce growth arrest and apoptosis in various human cancer cells by inhibiting the post-translational activation of Ras. FTase inhibitors also function to suppress the release of vascular endothelial growth factor (VEGF) from tumor cells by inhibiting Ras activation; however, the effects of FTase inhibitors on VEGF-induced angiogenesis in endothelial cells...

Journal: :Genetics 2012
Marsha D Wallace Adam D Pfefferle Lishuang Shen Adrian J McNairn Ethan G Cerami Barbara L Fallon Vera D Rinaldi Teresa L Southard Charles M Perou John C Schimenti

Identifying genomic alterations driving breast cancer is complicated by tumor diversity and genetic heterogeneity. Relevant mouse models are powerful for untangling this problem because such heterogeneity can be controlled. Inbred Chaos3 mice exhibit high levels of genomic instability leading to mammary tumors that have tumor gene expression profiles closely resembling mature human mammary lumi...

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