نتایج جستجو برای: liver protection

تعداد نتایج: 500048  

Journal: :American Journal of Transplantation 2004

Journal: :Journal of Immunology 2023

Abstract No highly efficacious anti-malaria vaccine exists, but whole parasite immunization (WPI)where hosts receive live attenuated Plasmodium parasites that arrest in the liver is only strategy confers complete protection against future infection. Liver-resident memory CD8 T cells (TRM)that express chemokine receptor, CXCR3, are essential for WPI-induced protection. Yet, whether CXCR3-depende...

صفاری, جواد , حیدریان, اسفندیار, قطره سامانی, کیهان, موحد محمدی, غزل,

Background and purpose: Acetaminophen is a routine analgesic and antipyretic agent that in overdose causes liver and kidney necrosis in both humans and animals. The cress (Lepidium sativum L.) contains flavonoid, alkaloid, and antioxidant components. In this study we investigated the hepatic protection of the hydroethanolic extract of cress against hepatotoxicity due to acetaminophen. Material...

2017
Zhengwei Leng Zhao Yang Songlin Hou Xiaoqiong Li Yunfeng Xiao Sisi Wang Lu Yang Guansheng Huang Jiali Li Jinrong Xia Zhengru Chen Siyu Li Zhonghui Zhou Jinghuang Chen Xia qinghua

To study the clinical manifestations, pathogenesis, and treatment of liver cirrhosis with chylous hydrothorax and ascites. Methods: A case of liver cirrhosis with chylous hydrothorax and ascites was reported and the related literature was reviewed. Results: The patient was hospitalized due to massive ascites. Paracentesis and a low-fat diet were effective. Diuretics, liver protection-therapy, a...

Journal: :Indian journal of experimental biology 2007
Sohini S V S Rana

Ascorbic acid treatment in arsenic trioxide treated rats increased arsenic excretion, inhibited lipid peroxidation, improved GSH status, regulated GSSG turnover and also restored glutathione-S-transferases activity in liver and kidney. Suitable mechanisms leading to ascorbic acid protection have been discussed. Upregulation of GSH dependent enzymes was found to be necessary for a protective eff...

Journal: :Best practice & research. Clinical anaesthesiology 2008
O Picker C Beck B Pannen

With recent advances in surgical and anaesthetic management, clinical medicine has responded to societal expectations and the number of operations in patients with a high-risk of perioperative liver failure has increased over the last decades. This review will outline important pathophysiological alterations common in patients with pre-existing liver impairment and thus highlight the anaestheti...

Journal: :Hepatology 1995
I Bohlinger M Leist J Barsig S Uhlig G Tiegs A Wendel

Mice sensitized with D-galactosamine (GalN) and challenged with recombinant murine tumor necrosis factor alpha (TNF alpha) developed severe apoptotic and secondary necrotic liver injury as assessed by histology, measurement of cytosolic DNA fragments, and determination of liver specific enzymes in plasma. Pretreatment with recombinant human interleukin-1 beta (IL-1) rendered mice insensitive to...

2014
Lili Ji Yibo Ma Zaiyong Wang Zhunxiu Cai Chun Pang Zhengtao Wang

Quercetin is a plant-derived flavonoid that is widely distributed in nature. The present study is designed to analyze the underlying mechanism in the protection of quercetin against pyrrolizidine alkaloid clivorine-induced acute liver injury in vivo. Serum transaminases, total bilirubin analysis, and liver histological evaluation demonstrated the protection of quercetin against clivorine-induce...

Journal: :World journal of gastroenterology 2010
Arthur I Cederbaum

S-adenosyl-L-methionine (SAM) acts as a methyl donor for methylation reactions and participates in the synthesis of glutathione. SAM is also a key metabolite that regulates hepatocyte growth, differentiation and death. Hepatic SAM levels are decreased in animal models of alcohol liver injury and in patients with alcohol liver disease or viral cirrhosis. This review describes the protection by S...

2015
Jessica A. Williams Wen-Xing Ding Natalia Osna Kusum Kharbanda

Alcoholic liver disease (ALD) is a major health problem worldwide, and alcohol is well-known to cause mitochondrial damage, which exacerbates alcohol-induced liver injury and steatosis. No successful treatments are currently available for treating ALD. Therefore, a better understanding of mechanisms involved in regulation of mitochondrial homeostasis in the liver and how these mechanisms may pr...

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