نتایج جستجو برای: fh protein

تعداد نتایج: 1237776  

Journal: :Journal of immunology 2010
Sarika Agarwal Sanjay Ram Jutamas Ngampasutadol Sunita Gulati Peter F Zipfel Peter A Rice

Neisseria gonorrhoeae can engage human complement receptor 3 (CR3) directly or through surface-bound iC3b. Factor H (fH) that binds to bacteria facilitates conversion of C3b to iC3b. fH also binds directly to CR3 on professional phagocytes. Certain nonprofessional phagocytes, such as primary cervical epithelial cells, also express CR3. We hypothesized that fH could bridge bacteria to CR3 and fa...

2012
Steven Johnson Lionel Tan Stijn van der Veen Joseph Caesar Elena Goicoechea De Jorge Rachel J. Harding Xilian Bai Rachel M. Exley Philip N. Ward Nicola Ruivo Kaushali Trivedi Elspeth Cumber Rhian Jones Luke Newham David Staunton Rafael Ufret-Vincenty Ray Borrow Matthew C. Pickering Susan M. Lea Christoph M. Tang

Neisseria meningitis remains a leading cause of sepsis and meningitis, and vaccines are required to prevent infections by this important human pathogen. Factor H binding protein (fHbp) is a key antigen that elicits protective immunity against the meningococcus and recruits the host complement regulator, fH. As the high affinity interaction between fHbp and fH could impair immune responses, we s...

Journal: :Infection and immunity 2002
Vinod Pandiripally Eugene Gregory David Cue

Opsonization of bacteria by complement proteins is an important component of the immune response. The pathogenic bacterium Streptococcus pyogenes has evolved multiple mechanisms for the evasion of complement-mediated opsonization. One mechanism involves the binding of human regulators of complement activation such as factor H (FH) and FH-like protein 1 (FHL-1). Acquisition of these regulatory p...

2010
Jonatan Leffler Andrew P. Herbert Eva Norström Christoph Q. Schmidt Paul N. Barlow Anna M. Blom Myriam Martin

Apoptotic cells are opsonized by complement components such as C1q and C3b, which increases their susceptibility to phagocytosis. Soluble complement inhibitors such as factor H (fH) also recognize apoptotic cells to minimize the pro-inflammatory effects of downstream complement activation. We used four radiolabeled protein constructs that span different regions of the 20 complement control prot...

Journal: :The Journal of infectious diseases 2015
Dan M Granoff Isabella Costa Monica Konar Serena Giuntini Koen K A Van Rompay Peter T Beernink

BACKGROUND The meningococcal vaccine antigen, factor H (FH)-binding protein (FHbp), binds human complement FH. In human FH transgenic mice, binding decreased protective antibody responses. METHODS To investigate the effect of primate FH binding, we immunized rhesus macaques with a 4-component serogroup B vaccine (4CMenB). Serum FH in 6 animals bound strongly to FHbp (FHbp-FH(high)) and, in 6 ...

بنی طالبی, مهدی, شایسته , فاطمه, شیرانی, منوچهر, مبینی, غلامرضا, پروین, ندا , صفاری چالشتری, جواد , طاهرزاده فر رخشهری, مریم , قطره سامانی, کیهان , مدرسی, مهرداد , هاشم زاده, مرتضی,

چکیده: زمینه و هدف: کلسترول بالای خانوادگی (FH) یک اختلال اتوزوم غالب است که عمدتاً به علت جهش های ژن LDLR و ApoB-100ایجاد می شود. تاکنون اساس مولکولی FH بطور مفصل در بسیاری از جمعیت ها تشریح شده است ولی هنوز اطلاعات مولکولی اندکی در ارتباط با FH در ایران موجود است. هدف از این مطالعه بررسی فراوانی 3 جهش شایع ژنی آپولیپوپروتئین B-100 ((ApoB-100 در یک جمعیت ایرانی است. روش بررسی: در این مطالعه...

Journal: :Journal of immunology 2014
Christophe Fleury Yu-Ching Su Teresia Hallström Linda Sandblad Peter F Zipfel Kristian Riesbeck

Haemophilus influenzae is a Gram-negative human pathogen that resides in the upper respiratory tract. Encapsulated H. influenzae type b (Hib) and type f (Hif) are the most common serotypes associated with invasive disease. H. influenzae displays various strategies to circumvent the host innate immune response, including the bactericidal effect of the complement system. In this study, we identif...

اسدی, سمیه, بنی طالبی, مهدی, حاجی حسینی بغداد آبادی, رضا, شایسته, فاطمه, صفاری چالشتری, جواد, طاهرزاده قهفرخی, مریم, قطره سامانی, کیهان, مبینی, غلامرضا, ناظم, حبیب اله, هاشم زاده چالشتری, مرتضی ,

چکیده: زمینه و هدف: هایپرکلسترولمی فامیلی (FH) بیماری غالب اتوزومال است که عمدتاً بدلیل جهش در ژن گیرنده لیپوپروتئین با دانسیته کم (LDLR) ایجاد می شود. این مطالعه با هدف بررسی تغییرات ژن LDLR بیماران مبتلا به کلسترول بالای خانوادگی در استان چهارمحال و بختیاری انجام شد. روش بررسی: در این مطالعه توصیفی- آزمایشگاهی، 57 بیمار مشکوک به FH غربالگری و با استفاده از روش PCR-SSCP جهش در پروموتر و اگز...

Journal: :Clinical and vaccine immunology : CVI 2014
Peter T Beernink Jutamas Shaughnessy Heather Stefek Sanjay Ram Dan M Granoff

Neisseria meningitidis causes disease only in humans. An important mechanism underlying this host specificity is the ability of the organism to resist complement by recruiting the complement downregulator factor H (FH) to the bacterial surface. In previous studies, binding of FH to one of the major meningococcal FH ligands, factor H binding protein (FHbp), was reported to be specific for human ...

Journal: :Journal of clinical microbiology 2003
John V McDowell Emily Tran Duncan Hamilton Jill Wolfgang Kelley Miller Richard T Marconi

Some Borrelia species associated with Lyme disease bind the complement-regulatory protein factor H (fH), a process that may aid in immune evasion. In this report we demonstrate that some Borrelia species associated with relapsing fever bind fH, but not those associated with avian borreliosis and epizootic bovine abortion. Cell-bound fH was also found to mediate cleavage of exogenously supplied ...

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