نتایج جستجو برای: ischemic brain damage

تعداد نتایج: 743027  

Journal: :Frontiers in bioscience : a journal and virtual library 2007
Guangwei Wei Dongmin Wang Huafei Lu Sophie Parmentier Qing Wang S Scott Panter William H Frey Weihai Ying

Cumulative evidence has indicated a critical role of poly(ADP-ribose) polymerase-1 activation in ischemic brain damage. Poly(ADP-ribose) glycohydrolase (PARG) is a key enzyme in poly(ADP-ribose) catabolism. Our previous studies showed that PARG inhibitors, gallotannin (GT) and nobotanin B, can profoundly decrease oxidative cell death in vitro. Here, we tested the hypothesis that intranasal deli...

Journal: :Journal of Cerebral Blood Flow & Metabolism 1994

دارابی, شمسی, محمدی, محمدتقی, نوروززاده, علی,

Background and purpose: Overproduction of free radicals after brain ischemia exacerbates brain infarction and edema specifically during early reperfusion. The scavenging property of water-soluble fullerene derivatives is proven, so, the aim of this study was to evaluate the effects of fullerenol nanoparticles on brain infarction and edema, especially oxidative damages during reperfusion in an e...

Journal: :razavi international journal of medicine 0
shamsi darabi department of physiology and biophysics, faculty of medicine, baqiyatallah university of medical sciences, tehran, iran mohammad taghi mohammadi department of physiology and biophysics, faculty of medicine, baqiyatallah university of medical sciences, tehran, iran; associate professor of physiology, department of physiology and biophysics, faculty of medicine, baqiyatallah university of medical sciences, tehran, iran. tel: ++98-2182483419 zeinab sadat sobhani department of physiology and biophysics, faculty of medicine, baqiyatallah university of medical sciences, tehran, iran

conclusions our findings indicate that fullerenol nanoparticles decrease the brain infarction through enhancement of the sod activity during cerebral ischemia-reperfusion injury. results occlusion of mca induced considerable infarction and lesion in ischemic hemispheres of control ischemic rats (527 ± 59 mm3) in accompany with a decrease in the glutathione content (45%), and sod activity (29%) ...

Journal: :Journal of Cerebral Blood Flow & Metabolism 1991

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