نتایج جستجو برای: wilms tumor gene

تعداد نتایج: 1471282  

Journal: :Turk patoloji dergisi 2017
Doaa Al Ghamdi Nasir Bakshi Mohammed Akhtar

Teratoid Wilms tumor is a rare variant of Wilms tumor composed predominantly of well-differentiated epithelial and/or mesenchymal heterologous elements. Like the classical Wilms tumor, this variant may also occur as a renal mass or may be found in extra renal locations. This tumor may be treated effectively by surgical resection; however, it generally fails to respond to chemotherapy. A review ...

2012
So-Young Lee Kyu-Rae Kim Jung-Yeol Park Jae Y Ro

Wilms' tumor is one of the most frequent malignant neoplasms in childhood. Advances in treatment modalities such as the combination of chemoradiation therapy with surgery have enhanced overall survival. However, recurrence of Wilms' tumor is still a problem. In this case, a 28-year-old female had experienced intermittent abdominal pain, and the computed tomography scan showed a huge pelvic mass...

Journal: :Cancer research 1993
M J Coppes Y Ye R Rackley X L Zhao G J Liefers G Casey B R Williams

The molecular genetic events involved in the etiology of granulosa cell, Sertoli cell, and Leydig cell tumors are unknown. The expression of the Wilms' tumor suppressor gene WT1 in granulosa and Sertoli cells prompted us to analyze this gene for mutations in 11 granulosa cell tumors, three Leydig cell tumors, and one Sertoli/Leydig cell tumor. Although most of these tumors express WT1 mRNA, non...

Journal: :Cancer research 2005
Debra J Morrison Milton A English Jonathan D Licht

Wilms' tumor or nephroblastoma is believed to arise from embryonic nephrogenic rests of multipotent cells that fail to terminally differentiate into epithelium and continue to proliferate. The WT1 tumor suppressor gene, a transcription factor controlling the mesenchymal-epithelial transition in renal development, is mutated in 10% to 15% of Wilms' tumors. This potentially explains the disordere...

Journal: :F1000Research 2023

Wilms tumor (WT) is the most common renal malignancy seen in pediatric patients. Although lungs are site of metastasis Wilms tumor, non-malignant pleural effusion has been infrequently reported. Here, we report a case an eleven-year-old female who presented with abdominal mass and progressive breathlessness. On further evaluation, she was found to have right-sided ipsilateral massive eff...

برهانی, دکتر مرتضی, کوهساری , دکتر محمدرضا ,

ABSTRACT: DRASH Syndrome is a rare congenital disorder,and the triad of drash syndrome are progressive renal insufficiency ,male pseudohermaphroditism & Wilms tumor .All of the reported cases were sporadic and autosomal dominant inheritance was suspected in some references. We diagnosed a 32- month child with this syndrome in IRAN in 1993. He was presented with wilms tumor and also had male p...

2006
Rachel A. Altura Marcus Valentine Hao Li James M. Boyett Patricia Shearer Paul Grundy David N. Shapiro Thomas Look

Wilms' tumor, an embryonic renal neoplasm diagnosed primarily in young children, can occur in either a noninheritable (sporadic) or a familial form, with the latter presenting earlier and more often at bilateral sites. Although familial Wilms' tumor is thought to develop through inherited and acquired mutational inactivation of the two alÃ-elesof pre disposing tumor suppressor genes, only a sma...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2006
Rachael Natrajan Suzanne E Little Jorge S Reis-Filho Lara Hing Boo Messahel Paul E Grundy Jeffrey S Dome Toni Schneider Gordan M Vujanic Kathy Pritchard-Jones Chris Jones

PURPOSE The most well established molecular markers of poor outcome in Wilms' tumor are loss of heterozygosity at chromosomes 1p and/or 16q, although to date no specific genes at these loci have been identified. We have previously shown a link between genomic gain of chromosome 1q and tumor relapse and sought to further elucidate the role of genes on 1q in treatment failure. EXPERIMENTAL DESI...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2012
Aleksandra Bielen Gary Box Lara Perryman Lynn Bjerke Sergey Popov Yann Jamin Alexa Jury Melanie Valenti Alexis de Haven Brandon Vanessa Martins Vincent Romanet Sebastien Jeay Florence I Raynaud Francesco Hofmann Simon P Robinson Suzanne A Eccles Chris Jones

We have previously demonstrated an increased DNA copy number and expression of IGF1R to be associated with poor outcome in Wilms tumors. We have now tested whether inhibiting this receptor may be a useful therapeutic strategy by using a panel of Wilms tumor cell lines. Both genetic and pharmacological targeting resulted in inhibition of downstream signaling through PI3 and MAP kinases, G(1) cel...

Journal: :iranian journal of pediatric hematology and oncology 0
hossein ayatollahi department of hematology and blood bank, faculty of medicine, cancer molecular pathology research center, mashhad univerسازمان های دیگر: cancer molecular pathology research center mohammad hadi sadeghian department of hematology and blood bank, faculty of medicine, cancer molecular pathology research center, mashhad univerسازمان اصلی تایید شده: دانشگاه علوم پزشکی مشهد (mashhad university of medical sciences)سازمان های دیگر: cancer molecular pathology research center sepideh shakeri cancer molecular pathology research center, mashhad university of medical sciences, mashhad, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی مشهد (mashhad university of medical sciences) seyyede fatemeh shams cancer molecular pathology research center, mashhad university of medical sciences, mashhad, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی مشهد (mashhad university of medical sciences) maryam sheikhi cancer molecular pathology research center, mashhad university of medical sciences, mashhad, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی مشهد (mashhad university of medical sciences) mohammad alidadi cancer molecular pathology research center, mashhad university of medical sciences, mashhad, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی مشهد (mashhad university of medical sciences)

increment. it is categorized based on genetic aberrations. some of these genetic disorders can determine minimal residual diseases (mrd) and prognosis of aml patients. wilms tumor (wt1) over expression is found in aml patients. the aim of this study was to determine the frequency of wt1 over expression in aml pediatric cases in north -east of iran. materials and methods: this retrospective stud...

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