نتایج جستجو برای: phenylketonuria

تعداد نتایج: 2147  

Journal: :Molecular genetics and metabolism 2013
A Wiedemann B Leheup S-F Battaglia-Hsu P Jonveaux E Jeannesson F Feillet

In our phenylketonuria (PKU) cohort of 120 patients, we uncovered a couple of cases of undiagnosed mild phenylketonuria (mPKU)/hyperphenylalaninemia (mHPA) in maternal parents of the PKU cohort. This finding prompted us to evaluate the risk of either mild phenylketonuria or mild hyperphenylalaninemia in the parent population whose children were diagnosed with hyperphenylalaninemia (HPA). Taking...

Journal: :The Turkish journal of pediatrics 2016
Betül Mazlum Banu Anlar H Serap Kalkanoğlu-Sivri Kader Karlı-Oğuz Şeniz Özusta Fatih Ünal

Phenylketonuria is one of the most prevalent autosomal recessive hereditary disorders in Turkey. If untreated, it results in severe brain damage and can also be associated with autism in certain patients. We present a three-year old boy who exhibited the symptoms of autism and was subsequently diagnosed with phenylketonuria. This case illustrates that because the majority of autism cases are id...

Journal: :Cell transplantation 2012
X Stéphenne F G Debray F Smets N Jazouli G Sana T Tondreau R Menten P Goffette F Boemer R Schoos S W Gersting M Najimi A C Muntau P Goyens E M Sokal

Phenylketonuria is a metabolic disease caused by phenylalanine hydroxylase deficiency. Treatment is based on a strict natural protein-restricted diet that is associated with the risk of malnutrition and severe psychosocial burden. Oral administration of tetrahydrobiopterin can increase residual enzyme activity, but most patients with severe clinical phenotypes are nonresponders. We performed l...

بینافر, سیما, مهدیه, نجات,

Background and purpose: Phenylketonuria (PKU), a genetic disorder with an autosomal recessive pattern of inheritance, is mainly due to phenyalanine hydroxylase deficiency. In Iran, many studies have investigated the genetics of this disease among different populations. This study aimed to report the frequencies of the mutations for each population as determined in different studies. Material...

Journal: :Molecular genetics and metabolism 2002
Linda L McCabe Bradford L Therrell Edward R B McCabe

Newborn screening has existed for approximately four decades. During that period of time, newborn screening has evolved conceptually from a laboratory test for a single disorder, phenylketonuria (PKU), to a multi-part public health system involving education, screening, diagnostic follow-up, treatment/management, and system evaluation. At a time when newborn screening is recognized as a model f...

Journal: :Pediatrics 2003
Susan E Waisbren Colleen Azen

OBJECTIVE To assess cognitive and behavioral outcome in treated maternal phenylketonuria (PKU) offspring. METHODS In this prospective, longitudinal study, 228 children who were born to mothers with treated PKU or untreated mild hyperphenylalaninemia were compared with 70 control subjects at 7 years of age. RESULTS Offspring cognitive outcome negatively correlated with the number of gestatio...

Journal: :Journal of medical genetics 1995
P Guldberg K F Henriksen I Sipilä F Güttler A de la Chapelle

The incidence of phenylketonuria (PKU) in Finland is extremely low, probably below 1 in 100,000. We describe the mutations and haplotypes in all four presently known patients. Mutation R408W was found on four mutant chromosomes (all haplotype 2), and IVS7nt1, R261Q, and IVS2nt1 were each found on a single chromosome. No mutation was found on the remaining chromosome. These findings support a pr...

2015
Elise Jeannesson-Thivisol François Feillet Céline Chéry Pascal Perrin Shyue-Fang Battaglia-Hsu Bernard Herbeth Aline Cano Magalie Barth Alain Fouilhoux Karine Mention François Labarthe Jean-Baptiste Arnoux François Maillot Catherine Lenaerts Cécile Dumesnil Kathy Wagner Daniel Terral Pierre Broué Loïc de Parscau Claire Gay Alice Kuster Antoine Bédu Gérard Besson Delphine Lamireau Sylvie Odent Alice Masurel Jean-Louis Guéant Fares Namour

BACKGROUND Mutations in Phenylalanine Hydroxylase (PAH) gene cause phenylketonuria. Sapropterin (BH4), the enzyme cofactor, is an important therapeutical strategy in phenylketonuria. However, PAH is a highly polymorphic gene and it is difficult to identify BH4-responsive genotypes. We seek here to improve prediction of BH4-responsiveness through comparison of genotypes, BH4-loading test, predic...

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