نتایج جستجو برای: neuronal cell death

تعداد نتایج: 1936424  

Journal: :Frontiers in Molecular Medicine 2023

Many neurodegenerative diseases fall under the class of known as proteinopathies, whereby structure and localization specific proteins become abnormal. These aberrant often aggregate within cells which disrupts vital homeostatic physiological cellular functions, ultimately contributing to cell death. Although disease research is typically neurocentric, there evidence supporting role non-neurona...

Journal: :Journal of Cell Biology 2006

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 1998
J Busser D S Geldmacher K Herrup

Alzheimer's disease (AD) is a major dementing illness characterized by regional concentrations of senile plaques, neurofibrillary tangles, and extensive neuronal cell death. Although cell and synaptic loss is most directly linked to the severity of symptoms, the mechanisms leading to the neuronal death remain unclear. Based on evidence linking neuronal death during development to unexpected rea...

Background and Objectives: Alzheimerchr('39')s disease (AD) is a neurodegenerative disease which is marked by impaired cholinergic function and decreased nicotinic acetylcholine receptor (nAChRs) density. nAChRs are important mediators of cholinergic signaling in modulation of learning and memory function. In Alzheimer hippocampus is particularly vulnerable to specific degenerative processes an...

Journal: :Journal of neurochemistry 2005
Agnieszka Kinsner Valentina Pilotto Susanne Deininger Guy C Brown Sandra Coecke Thomas Hartung Anna Bal-Price

In this study we investigated the mechanisms of neuronal cell death induced by lipoteichoic acid (LTA) and muramyl dipeptide (MDP) from Gram-positive bacterial cell walls using primary cultures of rat cerebellum granule cells (CGCs) and rat cortical glial cells (astrocytes and microglia). LTA (+/- MDP) from Staphylococcus aureus induced a strong inflammatory response of both types of glial cell...

  Background and Objective: As apoptotic cell death is extremely involved in physiological development and many pathological situations such as cancer and neurodegenerative diseases, the understanding of its molecular machinery can be useful in designing new therapeutic strategies. The present study investigated the temporal expression of the proapoptotic protein Bax in adult spinal motoneuron...

Journal: :Autophagy 2016
Cuicui Xie Vanessa Ginet Yanyan Sun Masato Koike Kai Zhou Tao Li Hongfu Li Qian Li Xiaoyang Wang Yasuo Uchiyama Anita C Truttmann Guido Kroemer Julien Puyal Klas Blomgren Changlian Zhu

Perinatal asphyxia induces neuronal cell death and brain injury, and is often associated with irreversible neurological deficits in children. There is an urgent need to elucidate the neuronal death mechanisms occurring after neonatal hypoxia-ischemia (HI). We here investigated the selective neuronal deletion of the Atg7 (autophagy related 7) gene on neuronal cell death and brain injury in a mou...

Journal: :Journal of neurophysiology 2010
Yongfu Wang Janna V Denisova Ki Sung Kang Joseph D Fontes Bao Ting Zhu Andrei B Belousov

N-methyl-D-aspartate receptors (NMDARs) play an important role in cell survival versus cell death decisions during neuronal development, ischemia, trauma, and epilepsy. Coupling of neurons by electrical synapses (gap junctions) is high or increases in neuronal networks during all these conditions. In the developing CNS, neuronal gap junctions are critical for two different types of NMDAR-depend...

2013
In-Young Choi Ji-Hyae Lim Chunsook Kim Hwa Young Song Chung Ju Won-Ki Kim

N-methyl-D-aspartate (NMDA) receptor-mediated excitotoxicity is one of the major causes for neuronal cell death during cerebral ischemic insult. Previously, we reported that the final product of lipid membrane peroxidation 4-hydroxy-2E-nonenal (HNE) synergistically increased NMDA receptor-mediated excitotoxicity (J Neurochem., 2006). In this study, we investigated the mechanism involved in the ...

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